Autism - Common observations by IsabellaThomas

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Original sender's name: Sheri Nakken Original sender's address: homeopathycures I found this to be an excellent summarySheri "IsabellaThomas" <isabella> Common observations. The purpose of this communication is to set out in simple terms the present status of the vaccine/autism problem. There is no evidence available to support the views held by a cabal of so-called medicinal product regulators and health care professionals that the observed increase in a regressive developmental behavioural condition having similarities to “autism” is not a real increase but is due to “better diagnosing” or “diagnostic redefining”. Since there is, after a decade of experience, no such evidence, the reality of an increase has to be accepted as fact. What follows has nothing to do with science, just common observation. Starting somewhere about 1995 reports began to appear concerning a hitherto unrecognised clinical condition in young children characterised by inflammatory bowel disease and a regressive behavioural disorder classified as autism. Since that time parental accounts have added numerous other abnormalities, all or some of which are present in affected children. The following is a list of reported characteristics- 1. A period of normal development2. Loss of previously learnt capabilities particularly speech, eye contact and, mistakenly, hearing.3. Appearance of worsening bowel problems - diarrhoea/constipation/incontinence/passage of undigested food4. Abdominal pain often severe and disabling5. Abnormal thirst6. Body temperature dysregulation and loss of thermal sensitivity7. Prolonged periods of hyperpyrexia in the absence of identifiable infection8. Sleep disturbance, frequently very distressing9. Repetitive muscular activity, apparently uncontrollable ­ bizarre posturing possibly due to pain10. Temper tantrums and prolonged screaming, convulsions11. Violent behaviour12. Even in the absence of speech the child may communicate distressing insight into their own disablilities13. Lack of appreciation of danger14. Dietary intolerance, often severe15. Failure to thrive16. Precocious sexual development17. Various biochemical abnormalities18. Endoscopic and biopsy evidence of serious bowel disease.19. Variable improvements with dietary restrictions20. Gait disturbance, unsteadiness and tip-toe walking21. Skin rashes in the absence of obvious cause22. Raised urinary uracil and thymine levels23. Dihydropyrimidine dehydrogenase deficiency This new syndrome has arisen within certain particular circumstances. The simple observational facts are as follows ­ 1. There were no reports of any affected children prior to the end of the 1980’s. Almost all have arisen in the past 10 to 15 years.2. There have been no reports of children suffering the initiation of this syndrome of abnormalities over the age of about five years and almost all have been at a still earlier age. There are no reports of the syndrome first appearing in adolescence or adulthood.3. This most unusual clinical picture has arisen simultaneously in the USA, the UK, Canada, Japan and most developed countries. That is, between about 1990 (probably later) and 2008. Whatever the cause of the syndrome may be it has to be consistent with these three incontrovertible facts. Any causative agent therefore must have been either absent or inactive prior to the late 1980’s, it must have been either inaccessible or inactive to individuals over the age of about five years and to have become active at precisely the same time over most of planet earth. This is not science, they are simply the requirements arising from common observation. It hardly needs to be said that causal possibilities are considerably restricted. A number of suggestions have been made which include pollutants, dietary factors, infectious agents and various toxic materials. None of these are compatible with the observed constraints which require, firstly, that they would have been absent or inactive prior to the end of the 1980’s, secondly, they would have to have been specifically and exclusively active in young children and, thirdly, they would have had to become available in widely separated countries at exactly the same time. Someone I know in the past has been a United Kingdom representative on the OECD Chemicals Programme, the EU toxicology programme, a medical advisor to the UK Ministry of Agriculture Fisheries and Food, the UK Health and Safety Executive and the Consumer Protection Unit of the Department of Trade and Industry and has known of no material or agent that would meet these requirements. The expansion of childhood vaccination programmes is a possible and plausible causative factor. This is not science, it is a fact which is consistent with the constraints. I fully agree that this is not proof of causality it is just the best possibility until someone comes up with a better one. Until that time we have to accept vaccination as the primary suspect. The list of characteristics and clinical disorders above strongly support the view that some or all are basically autoimmune conditions which are well known and accepted adverse reactions to most childhood vaccines. This raises yet another non-scientific question ­ if we accept that certain autoimmune conditions, Guillain-Barre syndrome, juvenile arthritis and type 1 diabetes mellitus for example, are vaccine related then why do we not accept these other autoimmune disorders in the same way? Finally, there are a number of children who, having suffered some or all of the listed disorders following a vaccination, undergo a modest improvement until the administration of a booster vaccination. Following a short period of time the child suffers an exacerbation of the original adverse effects ­ this is called positive rechallenge. In the absence of any other explanation the effect has to be accepted as causal. Isabella