Narcolepsy

[Guest guest]

Dear Marilette

 

Atma Namste,

 

Below is a mail received from one of our patients.

 

Can you please advise us the protocol.

 

With Love, Light and Peace

Diana

 

 

Quote

 

I have been suffering from sleep attack when I was

30-35 years old ( now I am 43) but I was not knowing

its a disorder, I have consulted the doctor when I

started experiencing the lose of grip, things used to

fall off my hand suddenly , he had asked me about the

sleep attack and halucination which was existing. He

concluded that the ailment is Narcolepsy and

priscribed medicines that was giving relief

temporarily.

 

I have noticed changes now in my losing grip and

halucination (almost gone) but sleep attack is in and

out like some days its perfect but most of the days

its really diffcult.

 

When I went through the web sites I realised mine is a

classic example of this disorder but this part of the

world its not known much.

 

Please help me to get out of this suffering.

 

With love, Light and peace

 

Manoj KV

 

Unquote

 

===================

 

 

Dear Diana,

 

Namaste.

 

Thank you for your email.

 

Medical Background:

 

Narcolepsy is a chronic neurological disorder caused

by the brain's inability to regulate sleep-wake cycles

normally. At various times throughout the day, people

with narcolepsy experience fleeting urges to sleep. If

the urge becomes overwhelming, patients fall asleep

for periods lasting from a few seconds to several

minutes. In rare cases, some people may remain asleep

for an hour or longer.

 

Narcoleptic sleep episodes can occur at any time, and

thus frequently prove profoundly disabling. People may

involuntarily fall asleep while at work or at school,

when having a conversation, playing a game, eating a

meal, or, most dangerously, when driving an automobile

or operating other types of potentially hazardous

machinery. In addition to daytime sleepiness, three

other major symptoms frequently characterize

narcolepsy: cataplexy, or the sudden loss of voluntary

muscle tone; vivid hallucinations during sleep onset

or upon awakening; and brief episodes of total

paralysis at the beginning or end of sleep.

 

In addition to daytime drowsiness and involuntary

sleep episodes, most patients also experience frequent

awakenings during nighttime sleep. For these reasons,

narcolepsy is considered to be a disorder of the

normal boundaries between the sleeping and waking

states.

 

For most adults, a normal night's sleep lasts about 8

hours and is composed of four to six separate sleep

cycles. A sleep cycle is defined by a segment of

non-rapid eye movement (NREM) sleep followed by a

period of rapid eye movement (REM) sleep. The NREM

segment can be further divided into stages according

to the size and frequency of brain waves. REM sleep,

in contrast, is accompanied by bursts of rapid eye

movement (hence the acronym REM sleep) along with

sharply heightened brain activity and temporary

paralysis of the muscles that control posture and body

movement. When subjects are awakened from sleep, they

report that they were " having a dream " more often if

they had been in REM sleep than if they had been in

NREM sleep. Transitions from NREM to REM sleep are

governed by interactions among groups of neurons

(nerve cells) in certain parts of the brain.

 

Scientists now believe that narcolepsy results from

disease processes affecting brain mechanisms that

regulate REM sleep. For normal sleepers a typical

sleep cycle is about 100 - 110 minutes long, beginning

with NREM sleep and transitioning to REM sleep after

80 - 100 minutes. But, people with narcolepsy

frequently enter REM sleep within a few minutes of

falling asleep.

 

 

The cause of narcolepsy remains unknown but during the

past decade, scientists have made considerable

progress in understanding its pathogenesis and in

identifying genes strongly associated with the

disorder. Researchers have also discovered

abnormalities in various parts of the brain involved

in regulating REM sleep that appear to contribute to

symptom development. Experts now believe it is likely

that-similar to many other complex, chronic

neurological diseases-narcolepsy involves multiple

factors interacting to cause neurological dysfunction

and REM sleep disturbances.

 

A number of variant forms (alleles) of genes located

in a region of chromosome 6 known as the HLA complex

have proved to be strongly, although not invariably,

associated with narcolepsy. The HLA complex comprises

a large number of interrelated genes that regulate key

aspects of immune-system function. The majority of

people diagnosed with narcolepsy are known to have

specific variants in certain HLA genes. However, these

variations are neither necessary nor sufficient to

cause the disorder. Some people with narcolepsy do not

have the variant genes, while many people in the

general population without narcolepsy do possess these

variant genes. Thus it appears that specific

variations in HLA genes increase an individual's

predisposition to develop the disorder-possibly

through a yet-undiscovered route involving changes in

immune-system function-when other causative factors

are present.

 

Many other genes besides those making up the HLA

complex may contribute to the development of

narcolepsy. Groups of neurons in several parts of the

brainstem and the central brain, including the

thalamus and hypothalamus, interact to control sleep.

Large numbers of genes on different chromosomes

control these neurons' activities, any of which could

contribute to development of the disease. Scientists

studying narcolepsy in dogs have identified a mutation

in a gene on chromosome 12 that appears to contribute

to the disorder. This mutated gene disrupts the

processing of a special class of neurotransmitters

called hypocretins (also known as orexins) that are

produced by neurons located in the hypothalamus.

Neurotransmitters are special proteins that neurons

produce to communicate with each other and to regulate

biological processes. The neurons that produce

hypocretins are active during wakefulness, and

research suggests that they keep the brain systems

needed for wakefulness from shutting down

unexpectedly. Mice born without functioning hypocretin

genes develop many symptoms of narcolepsy.

 

Except in rare cases, narcolepsy in humans is not

associated with mutations of the hypocretin gene.

However, scientists have found that brains from humans

with narcolepsy often contain greatly reduced numbers

of hypocretin-producing neurons. Certain HLA subtypes

may increase susceptibility to an immune attack on

hypocretin neurons in the hypothalamus, leading to

degeneration of neurons in the hypocretin system.

Other factors also may interfere with proper

functioning of this system. The hypocretins regulate

appetite and feeding behavior in addition to

controlling sleep. Therefore, the loss of

hypocretin-producing neurons may explain not only how

narcolepsy develops in some people, but also why

people with narcolepsy have higher rates of obesity

compared to the general population.

 

Other factors appear to play important roles in the

development of narcolepsy. Some rare cases are known

to result from traumatic injuries to parts of the

brain involved in REM sleep or from tumor growth and

other disease processes in the same regions.

Infections, exposure to toxins, dietary factors,

stress, hormonal changes such as those occurring

during puberty or menopause, and alterations in a

person's sleep schedule are just a few of the many

factors that may exert direct or indirect effects on

the brain, thereby possibly contributing to disease

development.

 

None of the currently available medications enables

people with narcolepsy to consistently maintain a

fully normal state of alertness. Thus, drug therapy

should be supplemented by various behavioral

strategies according to the needs of the individual

patient.

 

To gain greater control over their symptoms, many

patients take short, regularly scheduled naps at times

when they tend to feel sleepiest. Adults can often

negotiate with employers to modify their work

schedules so they can take naps when necessary and

perform their most demanding tasks when they are most

alert. The Americans with Disabilities Act requires

employers to provide reasonable accommodations for all

employees with disabilities. Children and adolescents

with narcolepsy can be similarly accommodated through

modifying class schedules and informing school

personnel of special needs, including medication

requirements during the school day.

 

Improving the quality of nighttime sleep can combat

EDS and help relieve persistent feelings of fatigue.

Among the most important common-sense measures

patients can take to enhance sleep quality are: (1)

maintaining a regular sleep schedule; (2) avoiding

alcohol and caffeine-containing beverages for several

hours before bedtime; (3) avoiding smoking, especially

at night; (4) maintaining a comfortable, adequately

warmed bedroom environment; and (5) engaging in

relaxing activities such as a warm bath before

bedtime. Exercising for at least 20 minutes per day at

least 4 or 5 hours before bedtime also improves sleep

quality and can help people with narcolepsy avoid

gaining excess weight.

 

Safety precautions, particularly when driving, are of

paramount importance for all persons with narcolepsy.

Although the disorder, in itself, is not fatal, EDS

and cataplexy can lead to serious injury or death if

left uncontrolled. Suddenly falling asleep or losing

muscle control can transform actions that are

ordinarily safe, such as walking down a long flight of

stairs, into hazards. People with untreated

narcoleptic symptoms are involved in automobile

accidents roughly 10 times more frequently than the

general population. However, accident rates are normal

among patients who have received appropriate

medication.

 

Finally, patient support groups frequently prove

extremely beneficial because people with narcolepsy

may become socially isolated due to embarrassment

about their symptoms. Many patients also attempt to

avoid experiencing strong emotions, since humor,

excitement, and other intense feelings can trigger

cataplectic attacks. Moreover, because of the

widespread lack of public knowledge about the

disorder, people with narcolepsy are too often

unfairly judged to be lazy, unintelligent,

undisciplined, or unmotivated. Such stigmatization

often increases the tendency toward self-imposed

isolation. The empathy and understanding that support

groups offer people can be crucial to their overall

sense of well-being and provide them with a network of

social contacts who can offer practical help and

emotional support.

 

Source - National Institute of Neurological Disorders

and Stroke, National Institutes of Health

 

Pranic Healing:

 

1. Invoke and scan before, during and after

treatment.

 

2. Instruct patient how to do pranic breathing. Ask

the patient to do 12 cycles of pranic breathing before

start of treatment; and continue during treatment.

 

3. After the patient has completed 12 cycles of

pranic breathing, apply general sweeping twice.

 

4. Localized thorough sweeping on the front and back

solar plexus chakra. Energize the heart chakra

through th eback heart chakra with LWG then with more

of ordinary LWV.

 

5. Localized thorough sweeping on the front and back

solar plexus chakra and the liver alternately with

LWG and LWO. Energize the solar plexus chakra with

LWB, LWG, LWO. Apply more localized thorough

sweeping.

 

6. Localized thorough sweeping with LEV or EV on the

entire head, the different sections of the brain:

left, right, front, back, middle center and base of

the brain. Rescan.

 

Apply more localized thorough sweeping until all the

sections of the brain are smooth and balanced.

 

7. Localized thorough sweeping and energizing on the

crown, back head minor, forehead, ajna, jaw minor and

throat chakras using LEV or EV.

 

 

8. Localized thorough sweeping on the entire spine

and both sides of the spine.

 

9. Localized thorough sweeping on the navel chakra

and basic chakra. Energize them with LWR.

 

10. Stabilize and release projected pranic energy.

 

11. Repeat treatment 3 times per week.

 

12. Instruct the patient how to do Superbrain Yoga properly.

This yoga may be done for 14 to 21 cycles per session

after each Pranic Healing treatment. On ordinary days, it

may likewise be practiced after physical exercise

for 1 to 3 sessions per day

to cleanse, energize and activate the different

sections of the brain to a certain degree.

 

Love,

 

Marilette

 

 

 

 

 

1. Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or if the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer.

 

2. Pranic Healers who are are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments. ~ Master Choa

Kok Sui

 

Miracles do not happen in contradiction to nature, but only to that which is

known to us in nature. ~ St. Augustine

 

Reference material for Pranic Healing protocols are the following books

written by Master Choa Kok Sui:

Miracles Through Pranic Healing, Advanced Pranic Healing, Pranic

Psychotherapy, Pranic Crystal Healing.

 

Ask or read the up to date Pranic Healing protocols by joining the group

through http://health./

 

MCKS Pranic Healing gateway website: http://www.pranichealing.org.