Jump to content
IndiaDivine.org

PART FOUR: Chernobyl and BSE?

Rate this topic


Guest guest

Recommended Posts

http://www.redflagsweekly.com/conferences/mad_cow/2004_jan13_4.html

 

 

AN ECODETECTIVE’S JOURNEY INTO THE CENTER OF NEURODEGENERATIVE DISEASE

 

By RFD Columnist, Mark Purdey

 

High Barn Farm, Elworthy, Taunton, TA43PX, UK.

Tel; 00 44 1984 656832.

MadCowPurdey

 

FINAL PAGE

Chernobyl and BSE?

But what about the more aggressive, new strain of TSE – the massive epidemic of

BSE / vCJD that suddenly blighted the UK’s young cattle and human population

post in November 1986? Did this big time epidemic result from an overexposure to

the artificial sources of the eco-prerequisites that we are talking about here?

 

In this respect, it seems likely that the UK’s BSE and vCJD epidemics were

caused by the simultaneous exposure of cattle, cats, humans and zoo animals to a

toxic combination of factors - the widely used copper-chelating organo

dithiophosphate (OP) insecticides (35) and the fall out of radioactive metals

from the Chernobyl eruption (20).

 

During the 1980s, British cattle herds and humans were exposed to exclusively

high doses of these insecticides for warble fly and head lice control

respectively (42), whereupon the prion proteins of the OP treated animals were

starved of their copper co partners (35). This rendered the prion protein

vulnerable to binding up with certain rogue replacement metals, such as the

radioactive strontium 90, which were rained down at high concentrations onto the

soils of NW Europe after the Chernobyl accident (20) – the precise region that

later became the world’s most intensive hotspot of BSE .

 

Political Perspectives.

 

The developed nations are only too keen to brandish the rogue states as

irresponsible for their clandestine development of nuclear and chemical weapons.

But they are not so keen to open the secret pages of their toxic history books

when it comes to answering their own public’s demands for data that may help

them understand the true causes of their ill health.

 

A substantial number of helpless human and animal populations were deliberately

subjected to high doses of radioactivity without their knowledge or consent. The

all too powerful politicians and scientific institutions who enacted these

atrocities have made damn certain that they can never be brought to account.

 

The only difference between the positions of the developed vis-a-vis the

undeveloped nations regarding their handling of weapons of mass destruction, is

that the less sophisticated rogue states have not yet developed a sufficiently

watertight infrastructure of secrecy and mass media spin to keep their various

acts of human and ecological barbarism under wraps. On the other hand, the

developed nations have successfully suppressed their shameful track record; and

in so doing they have committed further crime against humanity by deliberately

duping their populations with disinformation – a bibliography of bogus science

that has successfully misappropriated the cause of so many pollutant-induced

modern day ailments onto an assortment of genetic weaknesses, viruses, naturally

occurring toxins, or – as in the case of BSE - the sheep scrapie agent.

 

It seems that governments and corporations are deliberately conjuring up and

capitalising upon this phenomena of ‘natural’ scapegoats’ for their own

self-protection. It guarantees them a foolproof exemption from the flood of

compensation claims that could pop up if the catalogue of ill health effects

resulting from their bygone atomic antics and compulsory pesticide policies were

ever allowed to see the light of day.

 

If the western governments had permitted the toxic secrets of their atomic

backwaters to permeate the public domain, then we might be a lot further forward

in understanding the true causes of these neurodegenerative conditions today. If

you can understand the cause of a disease, then you are better equipped to work

out the best means of curing, controlling or preventing that disease - a cache

of knowledge that would prove extremely beneficial in the current world where

neurodegenerative disorders are beginning to reach epidemic proportions.

 

 

 

References

 

Fowler S., Plant toxin linked to Guam Dementia. New Scientist, 1987, August

13, p31.

Perl DP., ALS-Parkinsonism-dementia complex of Guam; In; Esire MM, Morris JH,

Eds, Neuropathology of Dementia, Cambridge University Press, Cambridge, p

268-292. 1997

Sacks O. The island of the colourblind and cycad island; Alfred Knopf, New

York, USA. 1997.

Celestial, Robert N. Perez WC; Teaching Ethics; Society for Ethics across

curriculum; Volume 3 (2) Spring 2003. Utah Valley State University, USA.

Sanderson, Vancil I. Sworn Statement. Tori Kae Nigro. Notary Public- State of

Nevada. Recorded in Washoe County. No 99-37452-2 August 30 2001.

‘Operation Crossroads 1946' DNA6032F; United States Atmospheric nuclear

weapons tests personnel review; human radiation experiments database: Operation

crossrodas 1946, Defence nuclear agency government pubs dept; released Sept 23

1984, page 142-143, 348-350, 444-447.

Donaldson LR, Seymour AH, Nevissi AE. University of Washington's

Radioecological studies in the Marshall islands. 1946-1977.

Frausto Da Silva, Williams RJP. The Biological chemistry of the elements 2rd

edition. Oxford University Press, Oxford. 2001.

Gajdusek DC., Foci of motor neurone disease in high incidence in isolated

populations of East Asia and the Western Pacific: in Rowland LP. Ed., Human

Motor Neurone Disease, New York, Raven Press p 363-393. 1982.

Bara M, Guiet-Bara A, Durlach J; 1993, Regulation of sodium and potassium

pathways by magnesium in cell membranes.Magnesium Res 6 (2) 167-177.

Free radicals in Biology and Medicine. Halliwell B, Gutteridge JMC, Eds 1989

2rd Edition. Clarendon Press, Oxford, UK

Cutler RG, Pederson WA, Camandola S, Rothskin JD, Mattson MP. Evidence that

accumulation of ceramides and cholesterol esters mediate oxidative stress

induced death of motor neurones in ALS, Annals of Neurol 52 2002.

Siddique T et al. Nature Genetics, October 3 2001.

Schwarcz R, Meldrum B. excitatory amino acid antagonists provide a

therapeutic approach to neurological disorders. The Lancet; 1985 July20th,

140-143.

Barium; Environmental health Criteria 107. International Programme on

chemical safety. WHO, Geneva.1990

Experimental and Clinical neurotoxicology; 2rd edition. Eds Spencer PS,

Schaumburg HH. Oxford Uni Press. 2000.

Kan M, Wang F, Kan M, To B, Gabriel JL, McKeehan WL. Divalent Cations and

heparin/Heparan Sulphate cooperate to control assembly and activity of the

fibroblast growth factor receptor complex. The American Society for

Biochemistry and Molecular Biology 1996, 271 (42) p 26143-26148.

Briscoe CLS. Blue Ribbon Panel Committee Action Report On Radioactive

Contamination in Guam Between 1946-1958; Ed; Castro WM. From the offices of

Senator Angel LG Santos and Senator Mark Forbes, Agana, Guam. November 12,

2002.

Miller WR, Sanzolone RF. Investigation of the possible connection of Rock and

soil geochemistry to the occurrence of high rates of neurodegenerative diseases

on Guam and a hypothesis for the cause of the diseases. Open-file report

02-475. US Dept of the Interior, US Geological Survey, Denver, CO 80225. 2002.

Eisenbud M, Gesell T. Environmental Radioactivity, 4th Edition. Academic

Press, London . 1997.

Gajdusek DC. Hypothesis: interference with axonal transport of neurofilament

as a common pathogenetic mechanism in certain diseases of the CNS. New England J

Med; 1985, 312 (11) 714-718.

Calne DB, Eisen A, McGeer E, Spencer P. Alzheimer's disease, Parkinson's

disease, and motor neurone disease: abiotrophic interaction between ageing and

environment. A Hypothesis. Lancet 1986 November 8th, 1067-1070.

Purdey M, BSE; Are we being fed a lie ? Ecologist, 2002, 32 (9) 33-37

Purdey M, Does an infrasonic acoustic shock wave resonance of the manganese

3+ loaded/ copper depleted prion protein initiate the pathogenesis of TSE.

Medical Hypotheses 2003 60 (6) 797-820.

Alldredge AW, Whicker WF, Hakonson TE. Retention of intravenously

administered Cs 134 in mule deer 10th Annual Progress report on AEC contract

AT(11-1)-1156.Dept Radiol Radiat Biol, Colorado State Uni, Fort Collins. p14-17,

1972.

Hakonson TE. Tissue distribution and excretion of Cs134 in the mule deer. MS

thesis. Colorado State University. Fort Collins pp121. 1967.

Alldredge AW, Lipscomb JF, Whicker FW. Forage intake rates of mule deer

estimated with fall out Cs-137. J Wildlife Management 38 508-516. 1974.

Whicker FW, Farris GC, Dahl AH. Concentration patterns of Sr 90, Cs 137, I

131 in a wild deer population and environment. Eds; Aberg B, Hungate FP.

Radioecological concentration processes. Pergamon Press, New York. p621-633

1966.

Hakonson TE, Whicker FW. Tissue distribution of radiocesium in the mule

deer. Health Physics, 1971, 21 (6) 862-866.

Arthur WJ 111. Plutonium intake by mule deer at Rocky Flats, Colorado. MS

Thesis. Colorado State Uni, Fort Collins, Co. pp123 1977.

Little CA. Plutonium in a grassland ecosystem. PhD thesis. Colorado State

Uni, Fort Collins, Co. pp170 1979.

Whicker FW, Farris GC, Remmenga EE, Dahl AH. Factors influencing the

accumulation of fallout Cs 137 in Colorado mule deer.Health Physics 1965, 11:

1407-1414.

Hiatt GS, Plutonium dispersal by mule deer at Rocky Flats, Colorado. MS

thesis. Colorado State Uni, Fort Collins. pp143 1977.

Arthur WJ, Alldredge AW. Soil Ingestion by mule deer in North Central

Colorado. Journal of Range Management 1979 32 (1) 67-71.

Purdey M, Ecosystems supporting clusters of sporadic TSEs demonstrate

excesses of the radical generating divalent cation , manganese, and deficiencies

of antioxidant co factors Cu, Se, Fe, Zn. Does a foreign cation substitution at

Prp's Cu domain initiate TSE ? Medical Hypotheses 2000 54 (2) 278-306

Spraker TR, Miller MW, Williams ES, Getzy DM, Adrian WJ, Schoonveld GG,

Spowart RA, O'Rourke KI, Miller JM, Merz PA. Spongiform encephalopathy in free

ranging mule deer, white tailed deer and rocky mountain elk in Northcentral

Colorado. J Wildlife diseases 1997 33(1) 1-6.

Dougherty J. EPA finds plutonium, dioxin in cement dust. Concrete Facts 1993

2 (4) 1-4 (PO Box 58, Laporte, Co 80535.

Williams ES, Young S. Chronic wasting disease of captive mule deer; a

spongiform encephalopathy. Journal of Wildlife Diseases; 1980, 16: 89-98.

Crom R, Cluster of CJD cases in Tucson. Report; The epidemic Intelligence

Service of the Arizona Department of Health Services, Pheonix. 1990.

Underhill PT. Naturally Occurring Radioactive Material. Advances in

Environmental Series. St Lucie Press, Florida. 1996

D'Alessandro M, Petraroli R, Ladogana A. Pocchiari M. High incidence of CJD

in rural Calabria. Lancet 1998; 352: 1989-1990

Purdey M, High dose exposure to systemic phosmet insecticide modifies the

phosphatidylinositol anchor on the prion protein; the origins of new variant

transmissible spongiform encephalopathy ? Medical Hypotheses 1998 50 (2) 91-111

Brown D, Prion and prejudice; normal protein and the synapse. Trends in

Neurosciences 2001 24 (2) 85-90.

 

 

 

 

 

 

 

Hotjobs: Enter the " Signing Bonus " Sweepstakes

 

 

Link to comment
Share on other sites

Join the conversation

You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.

Guest
Reply to this topic...

×   Pasted as rich text.   Paste as plain text instead

  Only 75 emoji are allowed.

×   Your link has been automatically embedded.   Display as a link instead

×   Your previous content has been restored.   Clear editor

×   You cannot paste images directly. Upload or insert images from URL.

Loading...
×
×
  • Create New...