Treatment of AIDS with Vitamin C

[Guest guest]

http://www.doctoryourself.com/aids_cathcart.html

 

Treatment of AIDS with Vitamin C

 

VITAMIN C IN THE TREATMENT OF ACQUIRED IMMUNE DEFICIENCY SYNDROME (AIDS)

Robert F. Cathcart III, MD

Medical Hypotheses, 14(4):423-433, Aug 1984.

Copyright ©, 1994 and prior years, Dr. Robert F. Cathcart. Permission granted

to distribute via the internet as long as material is distributed in its

entirity and not modified.

ABSTRACT

My previous experience with the utilization of ascorbic acid in the treatment of

viral diseases led me to hypothesize that ascorbate would be of value in the

treatment of AIDS (acquired immune deficiency syndrome). Preliminary clinical

evidence is that massive doses of ascorbate (50-200 grams per 24 hours) can

suppress the symptoms of the disease and can markedly reduce the tendency for

secondary infections. In combination with usual treatments for the secondary

infections, large doses of ascorbate will often produce a clinical remission

which shows every evidence of being prolonged if treatment is continued. This

clinical remission is achieved despite continuing laboratory evidence of helper

T-cell suppression. There may be a complete or partial destruction of the helper

T-cells during an initial infection that does not necessitate a continuing

toxicity from some source to maintain a permanent or prolonged helper T-cell

suppression. However, it is possible ascorbate may prevent that

destruction if used adequately during that prodrome period. Emphasis is put

upon the recognition and treatment of the frequent intestinal parasites. Food

and chemical sensitivities occur frequently in the AID syndrome and may

aggravate symptoms considered to be part of the AID syndrome. A topical C-paste

has been found very effective in the treatment of herpes simplex and, to a

lesser extent, in the treatment of some Kaposi's lesions. Increasingly, clinical

research on other methods of treating AIDS is being " contaminated " by patients

taking ascorbate.

INTRODUCTION

I had previously described that the amount of ascorbic acid which can be

tolerated orally by a patient without producing diarrhea, increases somewhat

proportionately to the toxicity of his disease (1,2,3,4). Among the roughly 80%

of persons who tolerate ascorbic acid very well, -bowel tolerance- will be

reached when in excess of 10 to 15 grams of ascorbic acid dissolved in water is

taken in 4 to 6 divided doses per 24 hours. The astonishing finding was that

when that same person is acutely ill with a mild cold, that tolerance may

increase to approximately 50 grams per 24 hours. A severe cold can increase

tolerance to 100 grams; an influenza, even up to 150 grams; and mononucleosis or

viral pneumonias, to as much as 200 grams per 24 hours. These higher doses may

have to be divided as frequently as hourly.

These large amounts of ascorbate are being drawn off the GI tract at a rate

sufficient to prevent significant amounts from reaching the rectum and producing

diarrhea. Measurements of ascorbate in urine, saliva, or serum indicate that if

sufficient doses of ascorbate are not given when a patient is ill, the body

level of vitamin C drops rapidly. In such a case, there is not enough vitamin C

left in the body, particularly in the cells directly involved by the disease, to

guarantee all the known housekeeping functions of the vitamin. Those functions

known to be dependent on vitamin C, including several metabolic reactions

necessary for proper functioning of the immune system, are put at risk of

malfunctioning. I call this condition -acute induced scurvy.-

PREMIERE FREE RADICAL SCAVENGER

The reason ascorbate ameliorates so many conditions is that it functions as the

-premiere free radical scavenger- (5). This function is not because it is the

most powerful free radical scavenger, but because it is possible to saturate

every cell of the body with more molecules of ascorbate than any other free

radical scavenger. The reason that it takes such massive doses for optimal

effect is because high concentrations of ascorbate must be driven into the cells

directly affected by the disease process sufficient to neutralize all of the

free radicals produced by that process, and have some left over for vitamin C

housekeeping functions. When a disease process involves free radicals, that

disease process is capable of being ameliorated by massive doses of ascorbate.

In the case of many infectious diseases, the relief from free radical

suppression of the immune system, allows for more effective attack on the

pathogen by that immune system.

-Note: this premiere free radical scavenger function has little to do with

nutrition but is a pharmacologic effect of ascorbate when utilized in unnatural

amounts for humans.-

Actually, the complete neutralization of free radicals requires several steps

involving other substances, e.g. glutathione. However clinically, the most

frequent limiting factor in the reduction of free radicals is ascorbate. In

certain conditions such as chemical allergies, certain other limiting factors

may become critically important, e.g. selenium and glutathione. Some have

worried that a buildup of dehydroascorbate would be toxic in certain of these

conditions. Clinically, this consideration has not created a problem when very

large doses of ascorbate are used. Perhaps it is the high ratio of ascorbate to

dehydroascorbate, I am careful to maintain in these patients, that protects

against any temporarily accumulating dehydroascorbate. Further, I should like to

point out that the dehydroascorbate formed should not be as toxic as that free

radical the ascorbate reduces as it itself is oxidized into dehydroascorbate.

In a way, it is unfortunate that this free radical scavenger and vitamin C are

the same substance. When ascorbate is destroyed in the process of destroying

free radicals, the vitamin C stores, particularly in the cells directly involved

in the disease process, are so depleted as to cause disorders of known

housekeeping functions of vitamin C.

It is certain that AIDS causes this depletion. The sicker the patient is, the

more ascorbate will be destroyed by the disease process. This depletion

certainly contributes to the terminal events and probably plays a key role in

the increased susceptibility of AIDS patients to various pathogens.

ASCORBATE VS. AN AIDS SUPPRESSOR FACTOR

A recent article describes the discovery of a -suppressor factor- in AIDS

patients. This suppressor factor was found to be neutralized in the test tube by

concentrations of ascorbate equivalent to that which would be achieved in a man

who ingested 10 to 20 grams of ascorbate a day. It was thought that this amount

was - " far too toxic " - to use in humans and that a less toxic antioxidant should

be found (6).

-Actually, 10 to 20 grams/24 hours of ascorbate is easily tolerated and is not

toxic- (1,2,3,4,7,8,9,10,11,12,13,14). Unfortunately, clinically I have shown

that the AIDS disease process destroys even larger amounts of ascorbate than the

10 to 20 grams because bowel tolerance is regularly increased to the range of

from 40 to 185 grams of C per 24 hours in the patient who has moderate Kaposi's

lesions and/or moderate lymphadenopathy. -Therefore, the 10 to 20 gram

equivalent of ascorbate in the test tube will not be adequate in vivo-.

PRELIMINARY STUDY

Because of the hypothesis that AIDS patients would benefit from large doses of

ascorbate, I began the actual treatment of AIDS patients and have found that

ascorbate is indeed very valuable when used in conjunction with certain

conventional treatments.

The following preliminary recommendations are based partly upon an anecdotal

group of approximately 90 AIDS patients who sought medical care from physicians

but who also took high doses of ascorbate on their own. Additionally, it is

based upon 12 of my AIDS patients, 6 of whom were given intravenous ascorbate

for a short period of time. Most of these patients have had considerable

improvement in their condition. This improvement seems somewhat proportional to

the amount of ascorbate taken by the patient relative to the severity of his

disease. If the patient tolerates enough ascorbate to " neutralize the toxicity "

of his disease and if the secondary infections are treated; his condition will

go into remission. Subjectively, symptoms decrease and increase inversely with

how closely the patient titrates to bowel tolerance.

The only death has been in a patient who had previously chemotherapy,

interferon, and total body Xray therapy. Additionally, his veins were so

destroyed by previous treatments that intravenous vitamin C therapy could not be

continued under the existing circumstances.

Such a preliminary report of recommendations is justified only because of the

urgency of the problem addressed and because in San Francisco and now New York,

news of the ascorbate treatment is spreading rapidly. Ascorbate is being used by

an increasing percentage of the AIDS patient population but without much

guidance. There have been many requests by physicians for the treatment

protocol.

ASCORBATE TREATMENT PROTOCOL FOR AIDS PATIENTS

The following protocol is recommended for AIDS and AIDS related conditions

including lymphadenopathy, idiopathic thrombo- cytopenia purpura, and

Pneumocystis carinii pneumonia.

As predicted, AIDS patients are usually capable of ingesting large doses of

ascorbate. It is desirable that the amount of ascorbate taken orally be

maximized. Patients are -titrated to bowel tolerance- (the amount that almost,

but not quite, causes diarrhea). A -balanced ascorbate- mixture is utilized

which is made up of a mixture of approximately 25% buffered ascorbate salts

(calcium, magnesium, and potassium ascorbate) and 75% ascorbic acid. This

mixture is dissolved in a small amount of water and taken at least every hour.

The purpose of the frequent doses and this balanced mixture is to maximize the

amount of ascorbate tolerated without producing diarrhea. Patients are permitted

to vary the percentage of ascorbate salts to straight ascorbic acid according to

taste. The usual amount tolerated initially is between 40 and 100 grams per 24

hours. -Doses in excess of 100 grams per 24 hours may be necessary with

secondary bacterial and viral infections-. As the patient's condition

improves, bowel tolerance will decrease.

When intravenous ascorbate is found necessary because the toxicity of the

condition exceeds the ability of the patient to take adequate amounts of

ascorbate to scavenge all of the free radicals created by the primary AIDS

infection and the various secondary infections, the following intravenous

solutions should be utilized. Sodium ascorbate buffered to a pH 7.4 and without

preservatives is added to sterile water in a concentration of 60 grams per 500

cc. This concentration is twice the concentration I have recommended before

because it is well tolerated in young males with large veins. Patients with

small veins may be best treated with solutions of 60 grams per liter. The time

of the infusions should be over at least a 3 hour period, preferably longer. As

much as daily administration of 3 bottles, 180 grams per 24 hours, may be

necessary in acutely ill patients, e.g. Pneumocystis carinii pneumonia,

disseminated herpes, disseminated cytomegalovirus, and atypical pneumonia.

Enough

ascorbate should be administered to detoxify the patient regardless of the

amount needed. Additionally, oral doses of ascorbate should be taken

simultaneously with the intravenous ascorbate. -Do not let the patients become

lazy and discontinue bowel tolerance doses of ascorbate while the intravenous

ascorbate is being administered-.

INTESTINAL PARASITES

If the AIDS patient has intestinal parasites, he must be treated for them. There

is a very high percentage of male homo- sexuals infected with intestinal

parasites. These intestinal parasites are themselves very immunosuppressive. The

prognosis for an AIDS patient is greatly enhanced by proper treatment of these

parasites. -Entamoeba histolytica-, especially, and -Giardia lamblia- must be

treated. Intestinal parasites, ordinarily considered -non-pathogens-, should be

treated. If negative, repeated stool examinations for ova and parasites should

be taken if there is the slightest clinical sign of intestinal parasite

infection. Samples should be fresh, not over 2 hours old. Laxatives may increase

chances of discovering the parasites. Additional samples may have to be taken

through a sigmoidoscope if other specimens are negative for ova and parasites.

With treatment, Herxheimer's reactions should be expected frequently. Symptoms,

including Kaposi's lesions, may be exacerbated, despite

the ascorbate, during treatment for intestinal parasites.

CANDIDA ALBICANS

Candida should be sought and treated. It should be emphasized to patients that

they owe it to themselves and society to treat the Candida consistently because

of the possibility of breeding resistant strains. The possibility of candida in

the gut, esophagus, mouth, sinuses, skin, etc. should be considered. In patients

who clinically appear to have Candida but in whom Candida cannot be cultured,

sensitivities to Candida should be suspected and treatment of especially the

bowel should be considered. Herxheimer's reactions, when antibiotics against

Candida are employed, should be considered one indication that Candida is a

problem. In these sensitive patients, foods and vitamins containing yeasts

should be avoided. Lactobacillus in large amounts should be fed to these

patients in an attempt to normalize bowel flora. Sugar and refined carbohydrates

should be avoided because Candida thrives on them.

There is a high incidence of food and chemical sensitivities associated with

Candida sensitivities (15,16,17) and Candida must be suspected whenever such

sensitivities are discovered.

FOOD AND CHEMICAL SENSITIVITIES

Food and chemical sensitivities, both IgE mediated allergies and enzymatic

deficiency allergies (EDAS), are common because of the disorders of the immune

system and the severe stress imposed by the AID syndrome. This increased

incidence of sensitivities may be associated with Candida, as discussed above,

but may also be a result of the AIDS infection. Rashes, edema, phlebitis, etc.

caused by corn, yeast (including yeast containing vitamins), molds, house gas,

automobile exhaust, certain herbal formulas, cosmetics, formaldehyde,

insecticides, paints, glues, and cigarette smoke have all been observed in my

small group of patients. Conditions such as Kaposi's lesions, lymphadenopathy

and probably idiopathic thrombocytopenia purpura, conditions which would

otherwise be considered just part of the AID syndrome or AIDS related, have been

seen to be aggravated by food and chemical sensitivities. These sensitivities

should be anticipated and offending substances should be removed from the

patient's diet and environment. Ascorbate may or may not block these

sensitivities significantly; however, ascorbate may decrease the intensity and

duration of the reaction in such a way as to make clinical discovery of the

offending substance easier.

This increased incidence of food and chemical sensitivities is very important to

understand because apparent adverse reactions to vitamin C may occur. These

reactions are almost never due to the ascorbate itself. Most ascorbate is made

from corn. Minute amounts of chemicals used in the manufacture of ascorbate may

remain. Residuals of these substances are almost invariably the cause of the

sensitivity reactions. Ascorbates made from sego palm or from tapioca and which

presumably are manufactured with some different chemicals, are often tolerated.

Different brands should be tried. It is almost always possible to find some

ascorbate that is tolerated. This sensitivity problem is very important to deal

with because patients frequently feel their life depends on taking adequate

amounts of ascorbate and they may be correct in this feeling.

Many times gastrointestinal discomfort and excessive gas can be alleviated by

changing to the sego palm ascorbate or changing brands of ascorbate.

OTHER CONSIDERATIONS

Bacterial infections should be treated with appropriate antibiotics but large

amounts of lactobacillus should be administered with foods if there is the

slightest tendency to Candida infections or sensitivities. Ascorbate

administration should be intensified during treatment for bacterial infections.

Intravenous ascorbate may be necessary.

Viral infections should be treated with intensification of the ascorbate

treatment. Intravenous ascorbate may become necessary.

Immunosuppressive therapy should not be utilized.

Sugar and processed foods, foods with chemicals, recreational drugs, cigarettes,

alcohol, etc. should be avoided. Obvious nutritional deficits should be sought

and corrected. Additional supplimentation with especially zinc and selenium may

be helpful.

All sharing of body fluids and fecal material should stop (18). Repeated

exposures, not only to possible AIDS infection, but to the secondary infections,

especially intestinal parasites and Candida should be avoided.

HELPER/SUPPRESSOR CELL RATIO

With this protocol, it may be anticipated that a large percentage of patients

will slowly go into an extended clinical remission. Patients must be on guard to

sense any impending infection, colds, etc. The patient should begin the

additional large frequent doses of ascorbate within minutes. At the dose levels

that have been possible under circumstances imposed, a slow improvement of the

total number of T-lymphocytes may occur but helper/suppressor cell ratios may

remain suppressed. It appears that ascorbate may assist the immune system, but

that in addition, there are mechanisms whereby ascorbate acts against pathogens,

especially viruses and bacteria by mechanisms which do not depend on the

T-cells. For this reason, I would suggest using the ascorbate portion of this

protocol on children who have to be permanently isolated from the slightest

exposure to infections (bubble babies).

MONITORING VALUE OF ASCORBATE " BURN "

Roughly to the degree that a patient clinically perceives himself to feel toxic

(the amount of malaise, fever, pain, how swollen the lymph nodes, how much

anxiety, etc.), the more ascorbic acid can be tolerated orally without it

producing diarrhea. The amount tolerated becomes a rough measurement of

something that represents the immediate toxicity of the condition. I use the

expression " 100 gram cold " to mean that at the peak of the cold a patient

tolerated 100 grams per 24 hours of ascorbic acid without diarrhea. In cases

where I am not sure what is causing an increased tolerance or if a person is

multiply ill with several secondary infections, I refer to the processes going

on which are using up the ascorbate as the " -burn-. " Note that the amount of

ascorbic acid tolerated is only a good measure of this burn if it is the amount

determined by titrating to " true " bowel tolerance, i.e., diarrhea caused by

ascorbic acid in a patient who otherwise tolerates ascorbate well; not limits

set by " too much gas " , " don't like the taste " , " stomach too acid " , etc.

The amount of this burn has some practical and prognostic values; e.g., a

patient with a burn much over 25-30 grams almost inevitably has something the

matter with him and a thorough diagnostic workup is indicated. A lover of one of

the AIDS patients had a burn of 100 grams. It was found that his

helper/suppressor T-cell ratio was 0.7 but he had no other sign of disease. Over

a 6 month period, the burn has dropped to 25 grams. AIDS has not been diagnosed

in this patient but there is good reason to suspect that he has a pre-AIDS

condition. The AIDS patient himself has had his burn drop from 125 grams to 35

grams. His lymphadenopathy has improved considerably.

AIDS POSSIBLY INVOLVING A PERMANENT OR PROLONGED LOSS OF T-HELPER CELLS

One patient who managed to eliminate all signs of Kaposi's lesions while taking

ascorbic acid had had his burn down to 15 grams a day for 6 months despite the

helper/supressor T-cell ratio remaining at 0.2. There had been some slight

increase in the absolute number of helper and suppressor cells. Previously

detected shedding of CMV (cytomegalovirus) had apparently stopped. This patient

had 3 Kaposi's lesions (diagnosed as Kaposi's sarcoma on biopsy) recur on the

right foot following a cold, herpes simplex, and influenza, all within a 2 month

period. The burn markedly increased, peaking at 185 grams per 24 hours. In 2

weeks time, the patient had managed to eliminate all signs of the lesions on the

foot. The ascorbate burn slowly has lessened; now 2 months later, the burn is at

25 grams and decreasing.

This case, plus the previous two cases, strongly suggest that the basic AIDS

infection, probably caused by a virus, is no longer active in these cases and

that subsequent ascorbate burns and various later manifestations of the AID

syndrome are caused by secondary and opportunistic infections. One is reminded

of the permanent damage of certain viral infections in association with certain

predisposing factors initiating an immune response to the beta cells of the

islets of Langerhans and causing juvenile-onset diabetes (19).

ASCORBATE AND THE POSSIBLE PREVENTION OF AIDS

Morishige has demonstrated the effectiveness of ascorbate in preventing

hepatitis B from blood transfusions (20). A similarity exists between AIDS and

hepatitis B. It has been my experience that patients treated with large doses of

ascorbate during the acute phase of hepatitis will not develop chronic

hepatitis. My experience with herpes simplex has been the same. Although

ascorbate is helpful to a degree with chronic viral infections, it is in the

treatment of acute viral diseases that it is most effective.

It is on this basis that I recommend that all persons who fear exposure to AIDS

and certainly anyone receiving blood trans- fusions or other blood products

which could in the most remote way have been obtained from an AIDS carrier, be

put on bowel tolerance doses of ascorbate.

CONTROLLED STUDIES OF OTHER SUBSTANCES [may be] CONTAMINATED WITH ASCORBATE

As a result of publications in periodicals concerned about the AID syndrome,

(21,22) a rapidly increasing number of AIDS patients in the San Francisco Bay

Area are taking large doses of ascorbate. The same practice is starting in New

York and elsewhere. I would suggest that physicians conducting controlled

experiments on interferon, and shortly with interleukin 2, be sensitive to the

fact that their patients are, and will be con- taminating the experiments with

massive doses of ascorbate. Statistical analysis of the results of such trials

will probably be valueless. Ascorbate has been contaminating cancer treatment

studies for some time as a result of orthomolecular literature (23,24,25). I

estimate that a significant increasing percentage of cancer patients in

California and other parts of the world are taking massive doses of ascorbate.

Most of these patients are hiding this fact from their oncologist.

BROADER PROBLEMS

The AID syndrome has not only become a major threat to the special groups

ordinarily affected but threatens to spread at least to some extent into other

groups. The increasingly large number of persons infected by the disease

increases the possibility of mutations which could alter the routes of

infection. Even without this possibility occurring, the large population of

immune suppressed persons comprises a major health hazard because of the large

pools of secondary infectious diseases generated. The large, growing pool of

intestinal parasites, heretofore present in the western world in only small

numbers, is one example of that problem.

POSSIBLE ELIMINATION OF THE AID SYNDROME

Practical considerations (lack of money and lack of hospital facilities) have

prevented me from administering the doses of ascorbate which I think might

-possibly- eliminate the probable viral infection initiating the AID syndrome. I

suggest that the helper/suppressor T-cell ratios should be carefully monitored

while at least 180 grams/24 hours of ascorbate is administered intravenously. At

the same time bowel tolerance doses of ascorbate should be taken orally. This

program should be followed over an extended period of time (minimum 2 weeks) to

find out if there is any direct effect on the process causing the AIDS.

I have preliminary evidence in one patient in which the above program was tried

that while the secondary problems were markedly suppressed by the ascorbate (7

lbs, 11 oz in 14 days) that the basic AIDS condition was not reversed. This case

plus the cases implying the permanent or prolonged suppression of the immune

system make it essential to treat the prodrome stages of AIDS with ascorbate.

If there is not a complete elimination of the basic AIDS process, bowel

tolerance doses of ascorbate and the rest of the described protocol will

probably have to be maintained for life.

My experience (1,2,3,4), and experience of other researchers

(10,11,12,13,14,20,26,27) is that acute self limiting viral diseases can be

reliably cured with massive doses of ascorbate. Viral diseases that have become

chronic seem to involve pathologic processes which are not quite as susceptible

to ascorbate but which nevertheless are ameliorated, sometimes seemingly cured.

It is hoped that funds will be made available for such a project.

C-PASTE

Herpes simplex lesions can usually be made to more rapidly heal or be prevented

at the outset by increasing the doses of oral ascorbic acid and the application

of C-paste. C-paste is made with either ascorbic acid or sodium ascorbate and

water applied directly to the skin and covered with a bandage. Frequently, one

application will suffice for herpes. Care should be taken not to irritate intact

skin too much in sensitive skin areas, especially under adhesive bandages.

Frequently applications to intact skin where the patient perceives an outbreak

is about to occur will completely abort the attack. Several applications may be

necessary to penetrate through the intact skin.

C-paste has also been useful on early Kaposi's lesions. It should be applied up

to 4 times a day. Alternatively, soaks of 20% sodium ascorbate or ascorbic acid

(1 gram per 5 cc of water) for 15-30 minutes, 4 times a day may be helpful. Be

careful not to irritate the skin too much even with these solutions. Keep

ascorbic acid out of the eyes; a 20% -sodium ascorbate- solution can be used in

the eyes with care.

KAPOSI'S LESIONS

Kaposi's lesions have been described as behaving like an infectious disease

closely associated with CMV (28). With ascorbate treatment, Kaposi's lesions may

be made to go away if the patient takes enough ascorbate and the patient is not

burdened by multiple opportunistic infections. In patients with multiple

problems, there tend to be outbreaks of the Kaposi's lesions associated with

colds, parasites, herpes, or emotional stress and particularly associated with a

letdown in the amount of C taken. Even in patients with multiple lesions,

individual lesions can frequently be seen to lose color and flatten with the

local application of ascorbate soaks.

CONCLUSIONS

Ascorbate does ameliorate the AID syndrome to a significant degree. I want to

emphsize, however, the absolute necessity of massive doses. Additionally, one

must avoid and treat oppor- tunistic infections. Multiple infections, lack of

understanding in the use of C, or inability to tolerate the doses prescribed,

all result in a poor prognosis. The success of treatments with ascorbate

entirely depends on consistent administration of C sufficient to neutralize the

free radicals produced by the various diseases.

The use of ascorbate is increasing in the male homosexual population of the San

Francisco Bay Area and spreading across the United States. It will be very

interesting to see if there are any otherwise unexplained decreases in the rate

of increase of new cases of AIDS and associated deaths starting in San

Francisco. The use of C is contaminating otherwise thought to be controlled

studies of other therapeutic measures. Other considerations plus the potential

application of ascorbate as part of the treatment of all infectious diseases,

makes the clarification of the usefulness of ascorbate to the medical profession

essential.

CAUTION

If these oral solutions are used over a long period of time, care should be

taken to keep them off the teeth by using a straw in order to avoid enamel

damage. Sickle cell anemia and G-6-PD deficiencies should be ruled out where

indicated. In any condition requiring prolonged administration of large amounts

of any nutrient, I would advise seeking the advice of a specialist to avoid

induced deficiencies in other nutrients.

 

REFERENCES

1. Cathcart, R.F. Clinical trial of vitamin C. Letter to the Editor, Medical

Tribune, June 25, 1975.

*2. Cathcart, R.F. The method of determining proper doses of vitamin C for the

treatment of disease by titrating to bowel tolerance. J. Orthomolecular

Psychiatry, 10:125-132, 1981.

*3. Cathcart, R.F. Vitamin C: titrating to bowel tolerance, anascorbemia, and

acute induced scurvy. Medical Hypotheses, 7:1359-1376, 1981.

*4. Cathcart, R.F. C-vitaminbehandling till tarmintolerans vid infektioner och

allergi. Biologisk Medicin, 3:6-8, 1983.

*5. Cathcart, R.F. Vitamin C function in AIDS. Current Opinion, Medical

Tribune, July 13, 1983.

*6. Laurence J. The mystery factor that's destroying immunity. American

Health, May/June 1983.

*7. Stone, I. The Healing Factor: Vitamin C Against Disease. Grosset and

Dunlap, New York, 1972.

*8. Pauling, L. Vitamin C and the Common Cold. W.H. Freeman and Company, San

Francisco, 1970.

*9. Pauling, L. Vitamin C, the Common Cold, and the Flu. W.H. Freeman and

Company, San Francisco, 1976.

*10. Klenner FR. Virus pneumonia and its treatment with vitamin C. J. South.

Med. and Surg., 110:60-63, 1948

*11. Klenner FR. The treatment of poliomyelitis and other virus diseases with

vitamin C. J. South. Med. and Surg., 111:210-214, 1949.

*12. Klenner, F.R. Massive doses of vitamin C and the virus diseases. J. South.

Med. and Surg., 113:101-107, 1951.

*13. Klenner, F.R. Observations on the dose and administration of ascorbic acid

when employed beyond the range of a vitamin in human pathology. J. App. Nutr.,

23:61-88, 1971.

*14. Kalokerinos, A. Every Second Child. Keats Publishing, Inc., New Canaan,

1981

*15. Truss, C.O. Tissue injury induced by Candida Albicans: Mental and

neurologic manifestations. J. Orthomolecular Psychiatry, 7,1:17-37, 1978.

*16. Truss, C.O. Restoration of immunologic competence to Candida Albicans. J.

Orthomolecular Psychiatry. 9,4:287-301, 1980.

*17. Truss, C.O. The role of Candida Albicans in human illness. J.

Orthomolecular Psychiatry, 10,4:228-238, 1981.

*18. Mavligit, G.M., Talpaz, M., Hsia, F.T., Wong, W., Lichtiger, B., Mansell,

W.A., Mumford, D.M. Chronic Immune stimulation by sperm alloantigens. JAMA,

251:237-241, 1984.

*19. Notkins, A.L. The Causes of Diabetes. Scientific American, 241,5:62-73,

Nov. 1979.

*20. Murata, A. Virucidal activity of vitamin C: Vitamin C for the prevention

and treatment of viral diseases. Proceedings of the First Intersectional

Congress of Microbiological societies, Science Council of Japan, 3:432-42, 1975.

*21. Cathcart, R.F. Vitamin C function in AIDS. Bay Area Reporter, p.18, Nov

17, 1983.

*22. Cathcart, R.F. Vitamin C treatment protocol for AIDS, Bay Area Reporter,

p.14-15, Jan 5, 1984.

*23. Cameron, E. and Pauling, L. Supplemental ascorbate in the supportive

treatment of cancer: Prolongation of survival times in terminal human cancer.

Proc. Natl. Acad. Sci. USA, 73:3685-3689,

1976.

*24. Cameron, E. and Pauling, L. The orthomolecular treatment of cancer:

Reevaluation of prolongation of survival times in terminal human cancer. Proc.

Natl. Acad. Sci. USA, 75:4538-4542,

1978.

*25. Cameron, E. and Pauling, L. Cancer and Vitamin C. The Linus Pauling

Institute for Science and Medicine, Menlo Park, 1979.

*26. Belfield, W.O., Vitamin C in treatment of canine and feline distemper

complex. Veterinary Medicine/Small Animal Clinician, pp. 345-48, Apr 1967.

*27. Belfield, W.O. and Zucker, M. How to Have a Healthier Dog. Doubleday &

Company, Inc., New York, 1981.

*28. Siegal, F.P. and Siegal, M. AIDS:The Medical Mystery. Grove Press, Inc.,

New York, 1983.

--

Robert F. Cathcart, MD.

Allergy, Environmental, Orthomolecular Medicine and Orthopedic Medicine

127 Second Street, Suite 4, Los Altos, California, USA

Telephone: 650-949-2822

Fax: 650-949-5083

 

 

 

 

AN IMPORTANT NOTE: This page is not in any way offered as prescription,

diagnosis nor treatment for any disease, illness, infirmity or physical

condition. Any form of self-treatment or alternative health program necessarily

must involve an individual's acceptance of some risk, and no one should assume

otherwise. Persons needing medical care should obtain it from a physician.

Consult your doctor before making any health decision.

Neither the author nor the webmaster has authorized the use of their names or

the use of any material contained within in connection with the sale, promotion

or advertising of any product or apparatus. Single-copy reproduction for

individual, non-commercial use is permitted providing no alterations of content

are made, and credit is given.

 

 

 

 

 

 

Hotjobs: Enter the " Signing Bonus " Sweepstakes