HIV treatment?

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" 121 "

 

Thu, 29 Apr 2004 23:18:30 -0700

HIV cure cover up, for drug profits!

 

 

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab finding points to potential new class of

HIV treatments. Johns Hopkins scientists have found that simply increasing

manganese in cells can halt HIV's unusual ability to process its genetic

information backwards, providing a new way to target the process's key driver,

an enzyme called reverse transcriptase.

 

Some of the recent data on HIV shows that the trace metal balance in the blood

and cells is directly linked to the cure for HIV. Metals change the viral

replication factors within cells, See Ref below.

 

Fluorides in the body bind up these trace metals and make them insoluble and not

available for cell functions. The manganese and reverse transcription factor is

the main principle for the HIV infection of T-cells.

 

It comes down the Govt. helping to cover up the main cause of HIV due to the

fluorides-metals effects and this helping to make drug companies huge profits

for making reverse transcription drugs that can be patented. The trace mineral

methods are too simple for patents and the reporting of this effect leads to

huge liability problems for DOE and its reckless fluoride-uranium operations.

 

Metals like gold and silver colloid compete for the fluorides in the blood and

complex with them to allow their rapid excretion as soluble salts, and this

process cuts down on the fluorides complexing with manganese and other essential

metals for the cellular functions.

 

The article below shows that the simple trace metal manganese blocks the key

infection method of reverse transcription in T-cells.

 

Take note that metals like manganese are directly linked to mad cow brain

effects. Perhaps this is the cow brain and immune response trying to protect

itself from viral invasion in a similar fashion?

 

Jim Phelps

 

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab finding points to potential new class of

HIV treatments

 

Johns Hopkins scientists have found that simply increasing manganese in cells

can halt HIV's unusual ability to process its genetic information backwards,

providing a new way to target the process's key driver, an enzyme called reverse

transcriptase. By measuring DNA produced by a related reverse transcriptase in

yeast, the Hopkins team discovered that higher than normal levels of manganese,

caused by a defective gene, dramatically lowered the enzyme's activity. The

scientists then proved that HIV's reverse transcriptase responds to manganese in

the same way.

 

Hopkins graduate student Eric Bolton determined that the defective gene is PMR1,

whose protein carries both manganese and calcium out of cells. Using special

yeast developed by others at Hopkins, he discovered that manganese stops reverse

transcriptase, the team reports in the April 26 issue of Molecular Cell.

 

" These results really point to a never-before-proposed way to try to stop HIV in

its tracks -- that simply manipulating concentrations of a metal, manganese, can

have a profound effect on reverse transcriptase, " says Jef Boeke, Ph. D.,

professor of molecular biology and genetics at the school's Institute for Basic

Biomedical Sciences. " We expect the human equivalent of PMR1 could be a good

target for developing new drugs against HIV. "