Prevalence Of Celiac Disease Increased in Cardiomyopathy

[Guest guest]

A backgrounder on the relationship between disease and wasting

disorders puts things in perspective.

 

Myopathies are part of a group of disorders that are marked by low

mitochondrial ATP output; this causes cell senescence and cell

wasting. What they call idiopathic (no cause) on the street is called

toxin load in the textbook.

 

A mitochondrial insufficiency is actually a systemic involvement that

shows up in " weak areas " first for lack of a better term. It's the

reason people often have more than one degenerative illness.

 

Mitochondrial impairment, it is taught in med school, is promoted by

toxins; this is why areas that are prone to irritation and toxin

assault anyway are usually the first to go, and because the liver is

the most able to recover it's the bowel, lung and kidney, the three

organs most exposed to toxin load next to the liver, that are fairly

common targets.

 

The solution is to reduce toxin load and promote mitochondrial

function; this involves in an approach that actively promotes

restoration and metabolic increase.

 

Symptoms of celiac may not be celiac. The same protein gluten occurs

on the outside of candida albicans cells and causes the same

symptoms; this is why the treatment for intestinal dysbiosis -- bad

bowel bacteria and candida -- usually dramatically reduces the celiac

issues. And part of the treatment is carb reduction anyway

 

Duncan

 

, " Raven " <NWRaven

wrote:

>

> Prevalence Of Celiac Disease Increased in Cardiomyopathy

>

http://www.mercola.com/1999/archive/celiac_disease_and_cardiomyopathy.

htm

> Researchers in Rome, Italy, report that the prevalence of celiac

disease is increased in patients with idiopathic dilated

cardiomyopathy.