Treating Diabetes With Enzymes: What We Know Now.

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Treating Diabetes With Enzymes: What We Know Now.

By: Dr. William Wong, ND, PhD.

Up to a year ago, for anyone asking if systemic enzymes could help lessen

the load of troubles that beset Type 1 diabetic patients, I would have told

them about lowering pancreatic inflammation, and possibly helping with lower

extremity circulatory issues. I would have never suggested that the use

of enzymes could decrease the need for insulin, increase energy or reverse the

seemingly myriad of things diabetics suffer from. Then we started getting

information from Type 1 patients that amazed even me and that have subsequently

sparked new research. Here are two typical case histories.

 

Case History #1:

A Type 1 diabetic Native American patient from Montana in his mid 40's, very

insulin dependent, with peripheral neuropathy in the lower extremities (LE's)

and presenting paresthesia as well in the upper extremities (UE's) radiating distally

to the hand. Peripheral Vascular Disease (PVD) in the LE's had already

caused several toes to be amputated.

Patient began taking therapeutic doses of fibrinolytic systemic enzymes

(Vitalzym). Within weeks, circulation was opened in his feet and lower

extremities. Skin there returned to a pink / flesh

colour. Remaining toes now have full circulation and

are no longer candidates for amputation. Lower extremity and upper

extremity pain became paresthesia (tingling and pins and needles), and as a

result is much more bearable.

The patient's insulin needs were decreased.

Case History #2:

86-year-old male Caucasian from Las Vegas history of Type 1 Diabetes for

over 50 years. One below the knee amputation (left side) already done due

to DVP, the other leg about to be amputated due to general lack of blood flow

and arterial blockage. Poor circulation body wide and a gray / white

pallor to the skin also body wide. Neurological pain was had at both

lower extremities. Urine flow beginning to flag as patients kidneys

became laden with scar tissue (Glomerulosclerosis). Patient was

highly insulin dependent. Above that the patient was

functionally blind in one eye from a Lasix procedure that had generated scar

tissue over the retina.

After several weeks of systemic enzyme (Vitalzym) use the patient first

noticed a lessening in his lower extremity neurological pain

(neuropathy). His skin colour in the remaining leg changed to rosy as

circulatory pathways were opening. Outer layer of whitish dead skin shed

off leaving what resembled a "body wide dandruff", exposing new pink /flesh

tone skin beneath. The existing leg became pink with blood flow, no

longer ulcered, no longer had ischemic pain and was saved from amputation.

Urine flow increased as fibrin was lysed (eaten away) from the

kidneys. If the urine was allowed to stand in the toilet a layer of tiny

bits of fibrin (component of scar tissue) in what resembled fiberglass floated

to the top. The fibrosis that had blinded one eye was lysed away and the

patient now has better than 20/20 vision in that eye. Most significantly,

the patients own insulin production has returned (thought to be impossible

under the auto immune theory of diabetic pancreatic destruction). He is

no longer insulin dependent. After medical testing the patient is no

longer considered diabetic at all and is off all medication.

Sound fantastic? It did to me, even as a Naturopath who expects nature

to do fantastic things. Diabetes is one of those diseases you never

expect patients to get better from. Even after several years of

working with systemic enzymes I had heard of some Type 2 patients improving

their energy and leveling off their sugar highs and lows but I had never

expected any form of improvement in Type 1 patients, the medical literature was

very clear. Once the immune system destroyed the insulin producing

portions of the pancreas, there was no getting those tissues to function

again! That medical "truth" has turned out to be merely a medical theory.

 

Lets take a look at the present understanding of the root causes of diabetes

and add our own conjectures based on what we have observed

clinically. We know from the present research work being done that

the root cause of diabetes is inflammation of the pancreas. How and why

this inflammation sets in we yet do not know. As we also know from the

physiology of trauma, inflammation breeds fibrosis or scar tissue. One

follows a chronic course of the other.

Fibrosis is also the culprit in the Peripheral Vascular Disease. In

this condition, fibrin plugs form in the micro circulation (tiny blood vessels)

forming blockages to full blood flow. Fibrin also forms the matrix for

arterial plaque. Inflammation of trauma to the inner lining of an artery

(intima), causes the traumatized or weakened section to shore itself up with

scar tissue. On the spider web of scar tissue fat, calcium and heavy

metals accrue forming what we know as arterial plaque. Once the

fibrosis blockages become extensive enough, the patient presents the signs of

PVD, which are cold extremities, intermittent caludication (pain on walking

from lack of oxygen supply to the tissues known as ischemia), non healing

ulcerations of the skin and eventual death of tissue creating gangrene leading

to amputation.

The high blood sugar levels had during diabetes damages the body's

organs. One of the first organs to be damaged are the nerves to the legs

and then the arms. Wherever the circulation is poorest the nerve damage

follows and radiating nerve pain is had

(neuropathy). The damage begins with, you guessed

it, inflammation and progresses with, you guessed it again,

fibrosis. It is this inflammation into fibrosis that seems to

be a recurring theme in diabetes.

For a moment lets do some education on orally administered systemic enzymes.

They have a 5 decade history of wide spread medical use in Germany, Central

Europe and Japan with over 150 million patients in Europe alone having

undergone enzyme therapy in the last 4 decades. There

are over 200 peer-reviewed studies proving the absorption, therapeutic action

and total lack of toxicity (no LD-50) of systemic enzymes. Their primary

action is anti-inflammatory, (though not through a COX 1 or Cox 2 action.

The enzymes instead "eat" pro inflammatory cytokines). The enzymes also

have a proven lysing action on all types of fibrosis and scar tissue leaving

normal or endogenous tissue entirely intact and un-bothered. This is due

to the body "tagging" excesses of fibrin as exogenous proteins. (The subject of

protein tagging and its discoverer won the Nobel Prize in biology in the late

'90's). Entering the key words: systemic enzyme, serrapeptase,

nattokinase, bromelain, pancreatin, papain, trypsin, chymo trypsin into the

search engine at Pub Med will bring up some of the current research on systemic

enzymes and their applications. A search in the "medical fields" section

of www.mucos.cz will show

abstracts of the extensive older research done with the first systemic enzyme

blends of the 50's and 60's. It has to be said that there is

nothing, no drug or substance, in either the allopathic medical world or in the

natural health world that can remove scar tissue but highly fibrinolytic

systemic enzymes.

Current thinking on diabetes is that the body's immune system attacks the

pancreas creating inflammation. This may be so. Further, the

current thinking is that the inflammation brings about the destruction of the

Islets of Langerhans and its Beta Cells, the places where insulin is

made. This may not be so. If the studies that are currently being

planned and executed further demonstrate what we are seeing clinically with

Type 1 patients on systemic enzymes, then this point will have to be re-thought.

Clinically most of the Type 1 patients have a significantly lower need for

insulin while some no longer need the insulin at all. This would

suggest that the Beta Cells and the Islets are not destroyed. I

conjecture that they are merely clogged by the fibrosis created by the

inflammation. Once the causative inflammation is reduced and once the

fibrinolytic action of the enzymes has eaten away the fibrosis and reopened the

channels, then what ever production the Islets can make can actually get into the

system.

I believe that the global (body wide) non-toxic, anti-inflammatory effects

of highly fibrinolytic systemic enzymes and the scar tissue eating effects of

the same enzymes are the reasons we are seeing the decrease in pancreatic

inflammation, decrease in diabetic neuropathy, in it's associated Peripheral

Vascular Disease, and the decrease in insulin dependence we are seeing

clinically in Type 1 patients. Let's see if the research further

verifies the observed findings and gives us more insight into the pathways of

action.

 

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