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A friend sent me this. I am interested in short and long term

practices that could aid in bone growth, or blood flow from a TCM

standpoint. Is it possible for one to assess the lack of flow without

expensive appointments.

 

" Hey Carlos,

 

Sorry to bother you again, I knew I was forgetting something. I

wanted to ask if you were familiar with anything that stimulated bone

growth other than calcium and maybe manganese. I broke my 5th

metatarsal and growth plate in my ankle and the doctor was concerned

about the healing rate because apparently they receive so little blood

flow. It's been 6 weeks and has made minimal progress, so anything I

can do to move this process along I'm going crazy with this low

activity lifestyle. "

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Chinese Traditional Medicine , " audi0warfare " <carlos wrote:

>

> A friend sent me this. I am interested in short and long term

> practices that could aid in bone growth, or blood flow from a TCM

> standpoint. Is it possible for one to assess the lack of flow without

> expensive appointments.

>

> " Hey Carlos,

>

> Sorry to bother you again, I knew I was forgetting something. I

> wanted to ask if you were familiar with anything that stimulated bone

> growth other than calcium and maybe manganese. I broke my 5th

> metatarsal and growth plate in my ankle and the doctor was concerned

> about the healing rate because apparently they receive so little blood

> flow. It's been 6 weeks and has made minimal progress, so anything I

> can do to move this process along I'm going crazy with this low

> activity lifestyle. "

>

 

There are probably alot of TCM ideas and forumals that would help, but

I am unfamiliar with them.

 

One thing he could check is something like his vitamin D level,

without it, then your body can't use the calcium. Something like 70%

of the adult american population is low on vitamin D. There are also

some other vitamins and minerals that are essential for calcium to

work from a western standpoint... I'm not sure that that is what you

were looking for though.

 

Hopefully someone else on the group will have some idea's about how

the TCM view and formula's work.

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--Thank you for your reply posting-

 

 

 

Yes, I was going to have her take/do the following.

 

 

 

Supplement:

 

 

 

_Alkaline Water

 

_Lemon/Cayenne Coldbroth

 

_Flax Oil

 

_Mangosteen Antioxidant Formula

 

_Vitamin E

 

_Coenzyme Q10

 

_Branched Chain Aminos

 

_B-Vitamins w folic acid

 

_Green Food Formula

 

_Zinc

 

 

 

Treatment:

 

_15 minutes in sun a day for vitamin-D synthesis of oils

 

_Local cold press: Two 12 minute intervals, 5x a day

 

 

 

Carlos

 

 

 

 

 

 

 

 

 

_____

 

Chinese Traditional Medicine [Chinese Traditional Medicine ]

On Behalf Of mrasmm

Saturday, April 28, 2007 4:21 PM

Chinese Traditional Medicine

[Chinese Traditional Medicine] Re: Bone Growth

 

 

 

Chinese Traditional Medicine@ <Chinese Traditional Medicine%40>

, " audi0warfare " <carlos wrote:

>

> A friend sent me this. I am interested in short and long term

> practices that could aid in bone growth, or blood flow from a TCM

> standpoint. Is it possible for one to assess the lack of flow without

> expensive appointments.

>

> " Hey Carlos,

>

> Sorry to bother you again, I knew I was forgetting something. I

> wanted to ask if you were familiar with anything that stimulated bone

> growth other than calcium and maybe manganese. I broke my 5th

> metatarsal and growth plate in my ankle and the doctor was concerned

> about the healing rate because apparently they receive so little blood

> flow. It's been 6 weeks and has made minimal progress, so anything I

> can do to move this process along I'm going crazy with this low

> activity lifestyle. "

>

 

There are probably alot of TCM ideas and forumals that would help, but

I am unfamiliar with them.

 

One thing he could check is something like his vitamin D level,

without it, then your body can't use the calcium. Something like 70%

of the adult american population is low on vitamin D. There are also

some other vitamins and minerals that are essential for calcium to

work from a western standpoint... I'm not sure that that is what you

were looking for though.

 

Hopefully someone else on the group will have some idea's about how

the TCM view and formula's work.

 

 

 

 

 

 

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Hello,

 

My brother, who is a licensed practitioner of TCM gave me the

following advice when I was diagnosed with Osteopenia... Stay on the

Six Flavored Tea Pills. The kidneys rule the bones, and since I had

been having kidney problems for years, when that was going better,

bone growth would follow. I am also eating a sheet of Nori (seaweed)

every day to supplement minerals. Hope this helps.

 

Chinese Traditional Medicine , " audi0warfare " <carlos wrote:

>

> A friend sent me this. I am interested in short and long term

> practices that could aid in bone growth, or blood flow from a TCM

> standpoint. Is it possible for one to assess the lack of flow without

> expensive appointments.

Link to comment
Share on other sites

Guest guest

~~ " audi0warfare " wrote:

Bone Growth

A friend sent me this. I am interested in short and

long term

practices that could aid in bone growth, or blood flow

from a TCM

standpoint. Is it possible for one to assess the lack

of flow without

expensive appointments.

 

" Hey Carlos,

 

Sorry to bother you again, I knew I was forgetting

something. I

wanted to ask if you were familiar with anything that

stimulated bone

growth other than calcium and maybe manganese. I

broke my 5th

metatarsal and growth plate in my ankle and the doctor

was concerned

about the healing rate because apparently they receive

so little blood

flow. It's been 6 weeks and has made minimal

progress, so anything I

can do to move this process along I'm going crazy with

this low

activity lifestyle. "

 

- -- -

 

 

g'day TCMers,

-ran across this the other day

-pretty comprehensive, altho'mention of the vital

importance of Magnesium is absent

cheers,

dar

 

 

http://www.alternativemedicine.com/common/news/store_news.asp?StoreNewsID=13402 & \

storeID=02AD61F001A74B5887D3BD11F6C28169

 

Bones of Contention

 

By James Keough

 

Experts devoted to bone health have proclaimed for

years that we face an osteoporosis crisis of epidemic

proportions. And they marshall an arsenal of

statistics to prove their point. The National

Osteoporosis Foundation says one in two women and one

in four men over the age of 50 will have an

osteoporosis-related fracture in their remaining

lifetime and that osteoporosis is responsible for 1.5

million fractures a year in the US—300,000 of the hip;

700,000 of the vertebra; 250,000 of the wrist; and

300,000 other. Ten million people in the US (80

percent of them women) are said to have the condition,

and almost 34 million are at risk because of low bone

mass. According to The Mayo Clinic on Osteoporosis

(Mayo Foundation, 2003), only a third of the people

who break a hip return to being as active as they were

before the fracture, and nearly another third wind up

in a nursing home permanently. More frightening, a

study in 2002 reported that as many as 20 percent of

hip fracture patients die within a year of their

injury.

 

Dire statistics like these, repeated without question

on TV and the Internet, and in various print media

over the past two decades, have created a climate of

fear among women in particular about what seems to be

their very high odds of succumbing to this “silent

crippler.” Is that fear warranted? Not really,

especially if you step back and look at how

osteoporosis became front page news and who stood to

gain from getting it there.

 

Creating a high-profile disease

 

Before the 1970s, osteoporosis was considered a rare

disease that affected people in extreme old age.

People learned they had it when they incurred what’s

called a fragility fracture—a break resulting from

slight impact to a bone that had become brittle and

had lost its strength and flexibility. Typically, a

fall might result in a broken wrist or, more

seriously, a hip, or someone might fracture a vertebra

or two lifting something. Break enough vertebrae in

the thoracic spine (actually they collapse on

themselves rather than fracture) and, over time, you

would develop a dowager’s hump—but that was rare, too

(and, if you look around, still is).

 

The perception of osteoporosis began to change in the

late ’70s and early ’80s. In a 1998 article in Nexus,

an Australian magazine that focuses on what the editor

calls “suppressed information,” Sherrill Sellman, ND,

author of Hormone Heresy: What Women MUST Know About

Their Hormones (Getwell International, 2000), links

this change to the widespread use of synthetic

estrogen for the symptoms and supposed problems of

menopause during the 1960s and ’70s, and to a 1975

report in the New England Journal of Medicine that

claimed “the risk of endometrial cancer increased 7.6

times in women using estrogen.” After another study

confirmed these findings later that same month,

estrogen sales, specifically of a popular brand called

Premarin, plummeted, despite a belated attempt to

rectify the problem by adding synthetic progesterone

to the estrogen (as occurs naturally in a woman’s

body) and renaming the new drug hormone replacement

therapy. According to Sellman, sales remained

depressed, leading Ayerst, the manufacturer of

Premarin, to hire a top public relations firm in 1982

to market osteoporosis to the public using TV, radio,

and magazine ads. An old woman with a dowager’s hump

was the campaign’s poster child. Prior to this effort,

says Sellman, 77 percent of women had never heard of

osteoporosis.

 

But the idea soon caught on. In 1984—two years after

the start of Ayerst’s marketing campaign—a group of

doctors founded the National Osteoporosis Foundation.

Its mission: Prevent osteoporosis and find a cure. In

1988, the first dual energy X-ray absorptiometry

(DEXA) machines made their debut with the promise of

predicting the risk of bone fracture by measuring bone

mineral density (BMD). And in 1994, the World Health

Organization (WHO) redefined osteoporosis as extremely

low bone density—2.5 standard deviations (SD) below

“normal,” which they set as the average peak bone mass

of healthy young white women. (The WHO definition also

mentioned the presence of at least two other risk

factors, but they got lost or at least underplayed in

drug company marketing.) At the same time, WHO

described a new precursor to osteoporosis, which it

christened osteopenia, as 1 to 2.5 SD below normal.

Together, these two definitions shifted the diagnostic

focus from brittle, weak bones that break easily to

low bone density. They also turned osteoporosis into a

woman’s disease, despite the fact that men incur a

third to half as many fractures as women and are more

likely to die because of them. According to Gillian

Sanson, author of The Myth of Osteoporosis (MCD

Century Publications, 2003), as the result of the new

definition, low bone density, just one of the many

risk factors for osteoporosis, suddenly became the

disease.

 

By the early ’90s, osteoporosis had become a major

symptom of menopause, but according to the Ayerst

campaign, one with a specific treatment: hormone

replacement therapy. Soon Premarin would become the

highest selling pharmaceutical of all time.

If this is starting to read like the ravings of

conspiracy theorists, consider the size and value of

the osteoporosis “market.” Based on the WHO

definition, the Swedish Council on Technology

Assessment in Health Care determined that more than 70

percent of all women over age 50 have either

osteoporosis (22 percent) or osteopenia (52 percent).

The National Osteoporosis Foundation’s 2005 annual

report calls osteoporosis a “major public health

threat for an estimated 44 million Americans, or 55

percent of the people 50 years of age or older.” That

adds up to lots of bone mineral density tests and lots

of pharmaceuticals. In 2005, physicians wrote 39

million prescriptions for bisphosphonates, including

22 million for Fosamax, which racked up $3.2 billion

in sales for the pharmaceutical giant Merck. And just

last year, Business Week Online estimated the global

osteoporosis market at $6 billion and said it was

growing at a rate of 25 percent a year. As Senator

Everett Dirkson so aptly mused, “A billion here, and a

billion there, and pretty soon you’re talking about

real money.”

 

Putting motives aside for a minute, all of this raises

the question, has osteoporosis been overhyped? Yes,

according to Mark Helfand, MD, MPH, MS, of Oregon

Health and Science University in Portland. In 2000,

the Washington Post quoted him as follows: “I think

even people who agree that osteoporosis is a serious

health problem can still say it is being hyped. It is

hyped.” Susan Brown, PhD, director of the Osteoporosis

Education Project, thinks so, too. But, she says,

“It’s difficult to have clear thinking when there’s

one financial interest that is so great.” So maybe we

can’t put motives aside. Marcelle Pick, OB/GYN NP,

cofounder with Christine Northrup, MD, and others of

the medical clinic Women to Women, makes the

connection clear on the clinic’s website: “I rarely

criticize the drug companies, but in this case I have

to say the publicity about osteoporosis is mostly

about profits, not about women’s health.” Or, as

Helfand put it for the Post, “Most of what you can do

to prevent osteoporosis later in life has nothing to

do with getting a test or taking a drug.”

 

Flies in the ointment

 

The trouble with the current definition of

osteoporosis, beyond the fact that it spreads too wide

a net and in the process sells billions of dollars of

medications to women who don’t need them, is that the

main premise is wrong. Low bone density alone does not

predict fractures. Study after study has demonstrated

this, and, Sanson tells us, at least five government

agencies in Canada, Australia, France, and Sweden have

come to the same conclusion. She cites a 1997 review

by the British Columbia Health Technology Assessment

Agency: “Even the most favorable reports on the

effectiveness of bone mineral testing reveal that BMD

testing does not accurately identify women who will go

on to fracture as they age.” In fact, Brown points out

that in several large studies, “almost 50 percent of

the people who fracture do not have severely low bone

density.” We now know, she goes on to say, that we

cannot determine fracture risk by bone density. “It’s

no more important than family history, and it’s not as

important as weight.”

 

Proponents of bone density testing claim that DEXA

machines are the gold standard for measuring bone

health: The tests are objective and hence rigorously

scientific, the argument goes. Unfortunately, the

logic doesn’t really hold up. The machines measure a

patient’s bone density against the average peak bone

mass of a healthy young Caucasian woman. That in

itself begs obvious questions: What if you’re not

Caucasian? Why should someone 50 or 60 years old (or

older) be compared to a 20-something-year-old? But a

more critical eye would zero in on this notion of peak

bone mass. Turns out, it’s not necessarily finite: It

varies with ethnicity, age, country (and even regions

within countries), and season—bone mass is lower in

the winter than in summer. These variables could all

be factored in to create a standard, but surprisingly

no accepted national or international standard exists

for peak bone mass. In fact, the manufacturers of the

DEXA machines determine the average peak bone mass

that their machines read as “normal.” This means that

bone density readings vary from brand to brand,

sometimes quite considerably, making any comparison of

their different test results meaningless. Some

manufacturers take a more conscientious approach to

establishing their peak bone mass standards than

others, but the opportunity to put a thumb on the

scale, so to speak, is pretty obvious. And apparently

it’s hard to resist.

 

Sanson points out that two large studies in the US and

Canada measured more diverse samplings of young people

to establish independent DEXA standards. In the US,

the third National Health and Nutrition Examination

Survey (NHANES III) found in 1995 that the peak bone

mass established from this survey was much lower than

those used by the manufacturers and using it “cut the

prevalence of osteoporosis, as defined by BMD, by more

than half.” The Canadian Multicentre Osteoporosis

Study (CaMOS) bore similar results in 2000: “They

found the actual prevalence of osteoporosis (as

defined by low bone density) to be 16 percent in women

and 5 percent in men, as opposed to the official

Canadian estimates of 50 percent and 12 percent,”

Sanson says. Studies in Turkey and the United Kingdom

produced strikingly similar readings. As Woody Allen

reputedly said, “No matter how cynical you are, it’s

hard to keep up.”

 

If we go back to the pre-WHO definition of

osteoporosis as a fragility fracture, then a true

measure of our osteoporosis epidemic would be the rate

of hip fractures, which cost $10.3 billion to $15.2

billion a year in the US alone and likely will top

$131.5 billion worldwide by 2050. The emotional,

psychological, and physical tolls simply can’t be

measured. Sanson calls attention to a National

Institutes of Health report finding that 80 percent of

women 75 or older would rather die than experience a

hip fracture that landed them in a nursing home.

 

One would assume that hip fracture rates have been

rising steadily as the crisis has deepened. But that’s

not the case. “It’s quite interesting that hip

fracture rates in this country really haven’t changed

much since the late 1950s,” says Brown. One would also

expect that the average age of women with hip

fractures would have dropped over the past 20 years,

but that’s not the case either. In fact, most every

researcher or author who mentions it says the average

age stands around 79 or 80, and the Osteoporosis

Education Project says it’s 82 in New York City.

 

What has changed is the incidence of hip fractures as

a growing part of our population has aged. That makes

perfect sense, but the increase is linked to

demographics—age, diet, lifestyle, and more—not bone

density.

The truly startling thing about the number of hip

fractures is how much higher they are in the US and

Europe than in many undeveloped countries throughout

the world. According to Brown, hip fracture rates

worldwide vary 30- to 40-fold. But as countries in

Asia and elsewhere Westernized, hip fracture rates

increase, which has led Sellman to call osteoporosis

“a degenerative disease of Western culture.” If you

ignore the implicit moral judgment, osteoporosis is in

the same league as cardiovascular disease and type-2

diabetes, which everyone seems to agree are diet and

lifestyle related.

 

A new paradigm

 

We tend to think of the 206 bones in our bodies as

hard, almost inert things. Indeed the word skeleton

comes from the Greek word skeletos, meaning “dried

up.” Yet healthy bones are anything but dry, and

they’re never uniformly hard. Instead, our bones are

living organisms that, once they stop growing in size,

undergo constant repair and maintenance in a process

called remodeling. Two types of cells, osteoclasts and

osteoblasts, perform this continual bone improvement

project. The osteoclasts seek out old or injured bone

and then produce an acid to dissolve it into its

constituent parts—calcium, magnesium, and other

minerals. This signals the osteoblasts to begin making

new replacement bone, a much slower process that takes

anywhere from three to 18 months to complete,

depending on one’s age.

In our youth, the osteoblasts outperform the

osteoclasts, and our bones and bone mass grow.

 

In Strong Women, Strong Bones (G.P. Putnam’s Sons,

2000), Miriam E. Nelson, PhD, explains how our bone

mass doubles from birth to age 2 and then doubles

again by age 10 and again by the end of puberty so

that by age 18 we have about 90 percent of our peak

bone mass. After a period of relative equilibrium,

Nelson explains, the balance between osteoclast and

osteoblast activity shifts in our mid-30s (somewhat

later in men), and we start losing bone mass at the

rate of 0.5 to 1 percent a year.

 

Unfortunately for women, bone loss accelerates in the

years surrounding menopause to anywhere from 1 to 3

percent (and as high as 5 percent) a year. It tapers

off about five to six years after a woman’s last

period until age 70 when bone loss drops back down to

the rate before menopause. Depending on how high the

rate of loss gets and how long it occurs, a woman

could reach her late 80s or early 90s—the prime

fracture years—with just 50 percent of her peak bone

mass left.

 

Remember, though, that peak bone mass varies

considerably from woman to woman and it doesn’t

correlate to fragility fractures. Remember, too, that

it’s natural to lose bone mass as we age—our bodies

have operated this way for millennia. The medical

community has somehow forgotten this latter fact or

consciously ignored it in its urge to stop bone loss,

first with synthetic estrogen, which among other

things stimulates osteoblast activity and suppresses

osteoclasts, and then with anti-resorption drugs like

Fosamax and Actonel, which suppress osteoclast

activity. The problem with this approach? “You cannot

have buildup of bone without breakdown of bone; they

go together,” says Brown. If you suppress osteoclast

activity, you also suppress the osteoblasts. Brown

says of these drugs, “They actually bring premature

death to the bone breakdown cells.” (See “Bad to the

Bone” on page 67.)

 

A more holistic, and arguably more successful,

approach championed by the Osteoporosis Education

Project, Women to Women, and many other

forward-looking practitioners, tackles both sides of

the bone resorption equation by urging women to

eliminate those activities and foods that contribute

to bone loss and encourage those that build bone.

In addition to the natural aging process and our

genetic makeup, a number of lifestyle, medical, and

dietary factors contribute to the loss of bone mass.

Individually (and certainly combined), the following

can increase normal bone loss to critical levels.

 

• Smoking. According to Nelson, “women with a smoking

history have significantly lower bone density and are

much more likely to suffer fractures than those who

never lit up.” Smoking further decreases estrogen

levels, which drop considerably during and after

menopause.

• Alcohol consumption. Studies have given us myriad

reasons for the health benefits of drinking in

moderation, but they’ve also shown that drinking in

excess increases the risk of low bone density and

fractures. According to Nelson, alcohol suppresses

osteoblast activity. High intake is also associated

with bad nutrition and an increase in falls that

produce fractures.

• Caffeine. As little as 300 to 400 mg of caffeine a

day (two to four cups worth of coffee) doubles the

risk of hip fracture. Coffee needn’t be the main (or

even primary) source: Black or green tea, chocolate,

and sodas all contain caffeine.

• Weight loss. Strict dieting at any age can lead to

malnutrition, especially of the nutrients bones need

to stay strong and healthy. In postmenopausal women

weight loss is even more insidious. Those annoying

(and persistent) extra pounds that appear around a

woman’s midsection at that stage in her life serve a

critical need: The fat cells they contain produce

estrogen, one of the purposes of which is to stimulate

osteoblasts to produce new bone.

• Pharmaceuticals. While many of us benefit from

prescription drugs, not all of them are benign when it

comes to bone loss. Foremost among the most dangerous

are corticosteroids (like Prednisone), which are used

extensively to fight inflammation and skin irritations

and to relieve arthritis and asthma. A 1993 study in

Annals of Internal Medicine reported that steroids

cause an 8 percent reduction in bone mass within four

months of use. (Brown says that steroid use causes 20

percent of all osteoporosis.) Thyroid hormone therapy

for hypothyroidism and goiter can lead to loss of bone

calcium and increase the risk of fracture. This places

older women at risk because they’re much more likely

than men to have thyroid problems. And researchers

have linked antacids and, more recently, proton pump

inhibitors like Nexium, Prevacid, or Prilosec, to an

increased risk of hip fractures. They speculate that

these drugs increase bone loss by inhibiting stomach

acid and thus reducing mineral absorption.

• Digestive disorders. Celiac disease, Crohn’s,

colitis, and irritable bowel syndrome all interrupt

the body’s normal absorption of calcium and other

critical bone-building minerals.

• Stress. Our bodies respond to the high stress of

life today by engaging our fight or flight mechanism.

It’s a collection of involuntary responses the body

makes to perceived danger so we’ll be ready either to

take action or run away. In normal circumstances, say

a near miss involving a bus, it causes a hormone

called cortisol to surge through the body and then

diminish once the threat is gone. A more muted version

of that reaction occurs when we’re exposed to stress

on the job or in our time-constrained lives, but

because the stress doesn’t subside, neither does the

cortisol. The effect on our bodies and our bones is

insidious: High levels of cortisol suppress the immune

system, cause calcium to be released from the bones

into the bloodstream, and suppress hormones and glands

involved in bone remodeling.

• Endocrine disorders. Hormones produced by the

endocrine system—the thyroid, pituitary, and

parathyroid glands—play a crucial role in bone

remodeling. Problems occur when the thyroid produces

either too little or too much thyroxin or its hormone,

calcitonin, gets out of whack. Calcitonin and

parathyroid hormone (PTH) regulate calcium levels in

the blood and tell the body either to store excess

calcium in the bones or to release it from them.

• Acidosis. To stay in balance chemically, the body

needs to maintain a slightly alkaline blood pH level.

To do this it must continually buffer the acids that

get produced as we metabolize our food. This is a

highly complex and exquisitely choreographed dance,

but the upshot for bone health is as follows: The body

looks first for alkalizing buffer elements like

potassium compounds in the blood and various organs;

when those are exhausted, it turns to the bones, the

body’s great alkaline storehouse. And since

maintaining homeostasis is a life-and-death matter,

the body will sacrifice bone to bring serum pH levels

back to the alkaline side of the ledger. Under normal

circumstances, borrowing from the bones is standard

operating procedure, and the alkalizing mineral

compounds get replaced once the crisis passes. But if

we eat a highly acidic diet that’s rich in sugar,

animal protein, or refined carbohydrates—the standard

Western diet, in short—our bodies have to work

continually to find buffering minerals. The borrowing

never stops, and the loan never gets repaid.

 

Building healthy bones

 

Losing bone mass might be an inescapable aspect of

getting older, and fragility fractures are certainly a

hazard in old age, but bone loss needn’t lead to a

fracture. Susan Brown jokes that we could cut hip

fractures in half if we could get everyone to die five

years earlier. The obvious (and far more attractive)

alternative is to figure out how to keep people

strong. “The message to individuals,” she says, “is if

you want to live long, take care of your

infrastructure.”

 

The obvious first step is to find ways to avoid or

minimize the lifestyle bone robbers. Quit smoking,

drink in moderation, cut back on caffeine, reduce the

stress in your life (or practice meditation or yoga),

and stop trying to lose those last 5 pounds. You can’t

do everything on your own, however. Countering the

effects of prescription drugs and endocrine disorders

requires working with your healthcare provider to find

alternatives and root causes—attempting that by

yourself is simply too risky.

 

The second step involves getting off the couch and

into your exercise or yoga clothes. Your body works on

a demand and supply basis when it comes to bones—if

your muscles and tendons put stress on your bones,

your body will respond by strengthening them. Any

number of studies have demonstrated this, and many of

them have shown that you can build bone density/mass

through exercise at any point in your life—even folks

in their 80s and confined to wheelchairs improved

their bone density by lifting 2-pound weights several

times a week. But not every kind of exercise will do.

Only weight-bearing activities like lifting weights,

running, and certain types of yoga (think Downward

Dog, handstands, and other arm-balancing poses) stress

the bones enough to stimulate growth. Walking and

swimming, both excellent aerobic activities and great

for the heart, have little effect on bone health.

 

Commit to a weight-bearing exercise routine for an

hour two to three times a week. According to Sanson,

this can significantly slow or prevent bone loss—a

benefit, she says, equal to that achieved by those who

exercise daily.

And finally, you can make dietary choices that will

not only reduce acidosis-caused bone loss, but will

provide your body with the key nutrients it needs to

build bone.

 

Reducing acidosis could require revamping your entire

diet, and that could take a good deal of discipline.

You won’t have to eat the gustatory equivalent of a

hair shirt, but you will need to reduce the amount of

acid-forming foods, such as meat, dairy, sugars, and

most grains, in your diet and increase those with

alkalizing powers, such as most fresh fruits and

vegetables. Ideally you’ll find a balance that will

allow you to eat a healthy variety of foods that won’t

need buffering with minerals from your bones. You can

learn more about these types of dietary changes from

The Acid Alkaline Food Guide by Susan E. Brown, PhD,

and Larry Trivieri Jr. (Square One Publishers, 2006)

and Food and Our Bones by Annemarie Colbin, MA, CHES

(Plume, 1998).

 

The last component in building healthy bones involves

providing your body with what the Osteoporosis

Educational Project calls the 19 key nutrients. The

most important, surprisingly, turns out to be vitamin

D, not calcium. That contradicts conventional medical

wisdom and the best efforts of the dairy industry, but

there isn’t any proof that consuming more dairy

products or taking calcium supplements alone reduces

the risk of fracture. For years advertisers have told

us that milk builds strong bones, and even the

government’s new dietary guidelines urge anyone older

than 8 to drink three cups of low-fat or fat-free milk

a day or the equivalent in yogurt or cheese. But the

Nurses Health Study, which followed 72,000 women over

25 years, found that women who drank milk twice a day

were as likely to suffer a fracture as those who drank

it once a week.

 

A similar study for men, the Health Professional

Follow-up Study, failed to find a link between calcium

intake and fractures, and a Swedish study of more than

60,000 women produced the same results. There is,

however, one rather strong relationship: The countries

with the highest level of dairy consumption, like the

US and Sweden, have the lowest bone density measures

and the highest rates of hip fractures.

 

Got myth?

 

Your body still needs calcium, of course, but only in

a form it can use, and only with adequate vitamin D to

help absorb it. As Brown points out, “If you’re low on

vitamin D, you absorb 65 percent less calcium.” So get

tested for vitamin D deficiency, and either get

outside in the sun (15 to 20 minutes of full-body

exposure a day without sunscreen) or supplement with

vitamin D3, a natural form of the vitamin found in cod

liver oil—it’s twice as potent as synthetic vitamin D.

Other crucial nutrients include potassium, magnesium,

and vitamin K.

 

Once you get past all the hype and fear mongering

about osteoporosis, you’re still left with the disease

itself and a set of risk factors against which you can

measure your own likelihood of facing fragility

fractures as you age. Forward thinking healthcare

practitioners are trying to steer the medical

profession away from its reliance on DEXA tests and

drug therapy for everyone. “The new edge,” says Brown,

“is to sort out who’s really at risk and then to

decide on the various types of treatments for those

people.” James Keough is the manuscript editor of

Alternative Medicine.

 

Bad to the Bone

 

Women diagnosed with osteoporosis are invariably told

they need to start drug therapy to cut their risk of

fractures. All of the drugs in the medical arsenal

increase bone density and, according to the studies,

have some short-term benefit in reducing fractures.

However, most of the drugs slow down or halt bone

remodeling, the natural process the body uses to get

rid of old bone and replace it with new. The long-term

implications aren’t yet known, which should engender

caution, and most of the drugs have some serious side

effects. Here’s what you should know before you start

using them.Bisphosphonates. Despite their humble

beginnings as industrial chemicals used for corrosion

prevention and the manufacture of laundry soap and

fertilizer, bisphosphonates like Fosamax and Actonel

have become mainstays in the treatment of

osteoporosis. Called anti-

resorption drugs, they suppress the activity of

osteoclasts, the cells that dissolve old or injured

bone. And they seem to work. “They halt bone breakdown

by perhaps 90 percent,” says Susan Brown, PhD,

director of the Osteoporosis Education Project (OEP),

“but they also halt bone formation by that much.” A

number of studies have shown that bisphosphonates

increase bone density in almost all women who take

them and that they reduce fractures by 30 to 50

percent in people with a high risk of fracture. OEP’s

ongoing vitamin D analysis reports similar results

with adequate amounts of vitamin D, calcium, and other

key bone nutrients.

 

Critics say bisphosphonates increase bone density at

the expense of new bone, which is always stronger and

more flexible than old. “This explains the increase in

bone density that occurs in the first year or so of

use,” writes Gillian Sanson in the Myth of

Osteoporosis, “but [it] also explains the subsequent

plateau effect.” The question is, says Brown, “Are you

going to end up with bone that looks sturdy . . . but

is fragile because it’s bone that hasn’t renewed

itself?”

 

A partial answer may lie in a serious bisphosphonate

side effect called osteonecrosis of the jaw—a

condition in which part or all of the jaw bone dies.

First encountered in patients who received intravenous

doses of Fosamax, it has more recently surfaced in

people taking the tablet form for a long time. The

reported number of cases remains small, but a June

2006 article in Lawyers and Settlements, estimates

that up to 10 percent of the people who’ve taken

Fosamax are at risk.

 

Comparatively less serious side effects include

digestive reactions: heartburn, indigestion, nausea,

diarrhea, and inflammation or ulceration of the

esophagus. Hormone replacement therapy. What started

as a “cure” for menopause “disorders” has now been

repackaged as preventative treatment for osteoporosis

after synthetic estrogen was shown to cause

endometrial cancer. According to The Mayo Clinic on

Osteoporosis, HRT “effectively reduces bone breakdown

and may result in a 5 to 6 percent increase in bone

density in lumbar vertebrae over one to three years of

use.”

 

The safety of HRT took another blow when the Women’s

Health Initiative stopped its study in 2002 upon

finding increased risk of breast cancer, stroke, and

heart attack in women taking Prempro, a combined

estrogen/progestin pill. The study also showed,

however, that Prempro reduced risk of hip and other

fractures.

Other studies have shown that the bone density

increase derived from HRT fades quickly after women

stop taking the drug, returning to age-related levels

in as little as a few months. And therein lies the

dilemma: Short-term use provides only transitory

results, and long-term use is fraught with dangerous

side effects. Raloxifene. Marketed as Evista and

sometimes called a “designer estrogen,” this selective

estrogen receptor modulator (SERM) slows bone loss to

a degree similar to estrogen, and after three years of

daily treatment, it reduced vertebral fractures (but

not other types) by 36 percent.

While raloxifene appears to lack the more severe

negative effects of HRT, it still increases the risk

of blood clots threefold. It also can cause leg

cramps, hot flashes, leg swelling, and flu-like

symptoms. Teriparatide. Unlike bisphosphonates, which

suppress bone remodeling, parathyroid drugs like

teriparatide (Forteo) actually stimulate bone growth

in both the hard cortical layer of bone and the

softer, more flexible trabecular bone. According to

the Mayo Clinic, daily injections combined with

calcium and vitamin D increased spinal bone density in

women with osteoporosis and previous vertebral

fractures by 9 to 13 percent and reduced other

fractures by 35 to 54 percent.

There’s some question about the quality of the bone

that’s being grown, and about 10 percent of

teriparatide users develop hypercalcemia, so serum

calcium levels need monitoring. But more seriously,

the drug comes with a warning about the possibility of

osteosarcoma. In fact, the drug can only be taken for

two years because clinical trials found no incidence

of bone cancer in that time frame. Calcitonin. The

Mayo Clinic says calcitonin “may slow bone loss and

increase bone density modestly” by affecting bone

resorption, and studies have found that over a

five-year period it reduced risk of vertebral

fractures by 36 percent. Sanson says that it may have

an analgesic effect on pain from vertebral fractures.

Milder and seemingly safer than the other drugs, it is

also less effective. Taken as a nasal spray, it can

cause nasal dryness and irritation, back and joint

pains, and headaches

..

Before the Fall

 

What you can do to prevent hip fractures as you age

 

The conventional view of osteoporosis sees hip,

vertebral, and wrist fractures as the result of weak

bones—strengthen an individual’s bones (often with

medications), and the risk of fractures declines.

Nothing wrong with that approach, theoretically, but

there’s a simpler, faster, and less expensive way not

only to cut fracture risk, but to cut the absolute

number of osteoporotic fractures: Prevent falls. The

Centers for Disease Control and Prevention (CDC)

report that falls cause more than 95 percent of hip

fractures in adults 65 and older, which led to more

than 309,500 hospital admissions in 2003. That figure

represents a 19 percent increase over admissions in

1993. Looking at our aging population, researchers in

1990 predicted the number of hip fractures in the US

would surpass 500,000 by the 2040. How important is

fall prevention? A 2005 Swiss study of 7,788 women

aged 70 or older found that a high risk of falling

predicts a three times greater incidence of hip

fracture, leading a researcher to say that the risk of

falling may be more important than high calcium

intake.

 

Here’s how you can keep yourself or an elderly loved

one from experiencing a fracture-causing fall. Get

lots of exercise. Weight-bearing physical activity

like resistance training and some forms of yoga will

help build strong bones, but it also increases muscle

mass and strength.

That strength will help a person rise from a chair or

climb stairs more securely and will increase her

confidence. Other forms of exercise help improve

balance, a critical factor in avoiding falls. Studies

have shown that the slow, graceful movements of t’ai

chi measurably improve balance in people of all ages.

Check your meds. A number of prescription and

over-the-counter drugs have side effects that increase

the risk of falling, especially when more than one

drug is involved. Sedatives, antidepressants, and

antipsychotic drugs can reduce mental alertness,

interfere with balance and gait, and cause a sudden

drop in systolic blood pressure, any one of which

could lead to a fall.

 

Ask your physician to review all of your current

medications, including those nonprescription items you

use regularly, with preventing falls in mind. And

limit alcohol intake when you take medications. Get

your eyes examined. This may seem like a no-brainer,

but poor eyesight is a frequent cause of falls. Older

eyes can change quickly; a yearly visit to the

optometrist could save your hip. Reduce hazards in the

home.

 

Most falls occur in the “unsafety” of our own homes,

so removing potential culprits can go a long way

toward keeping us on our feet.

Among the CDC’s suggestions: Improve lighting; put

nonslip mats under throw rugs (or remove them

entirely); fix loose or uneven steps; fix loose

handrails (and consider putting one on both sides of

the stairs); and keep wires from lamps and electrical

appliances coiled up and close to the wall. Wear hip

protectors.

If you or someone you know is at high risk for a hip

fracture, think seriously about wearing hip

protectors. They won’t do a lot for your figure, but

they dramatically reduce hip fractures. A study in the

New England Journal of Medicine found that hip

protectors cut the rate of fracture by more than half

and may also reduce pelvic fractures. A 2000 study in

Advance for Physical Therapists and PT Assistants

notes that a group of frail, at-risk women who wore

hip pads fell without fracture five times more than

the healthier control group.

 

© 1999-2007 Living Naturally and Alternative Medicine

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For more info on a TCM formula that promotes osteoblasts, see post

#1615 in the message section. WARNING: You still need to check out the

TCM picture before using this or any TCM formula. There may be cases

where this is not an appropriate formula because of the total TCM

clinical picture.

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Chinese Traditional Medicine , " Shoshanah " <ositaguapa

wrote:

> The kidneys rule the bones, and since I had

> been having kidney problems for years, when that was going better,

> bone growth would follow.

 

I want to emphasize this for the beginning TCM students on the list.

Any time there are bone problems suspect and rule in or rule out

possible Kidney imbalance. The health of the Kidneys often are

reflected in the bones and the teeth.

 

I also want to emphasize that you'll run across cases where the

kidneys are OK according to allopathic standards, but the Kidneys are

severely imbalanced.

 

Bone problems are a general indicator of possible Kidney imbalance.

You'll need to pay attention to other signs and symptoms to narrow

the diagnosis down to which particular Kidney imbalance (if there is

one). Kidney Yang Deficiency, Kidney Yin Deficiency, Kidney Qi

Deficiency, Kidney Jing Deficiency, etc.

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Guest guest

Great info, thankyou Victoria, Shoshanah, Dar and Mrasmm!!

 

 

 

I don't want to dominate the discussion with on-going questioning, but how

could I deduce which one it is? Yin Yang, Qi, or Jing. Im guessing since

she has poor Qi flow its blood related. If I am asking too much.or if this

question is tired, I can search the databases, its just that the answers

here are so current, direct and thorough. I thought id try to did a little

deeper.

 

 

 

Thanks again!

 

 

 

Carlos

 

 

 

_____

 

Chinese Traditional Medicine [Chinese Traditional Medicine ]

On Behalf Of victoria_dragon

Sunday, April 29, 2007 12:47 PM

Chinese Traditional Medicine

[Chinese Traditional Medicine] Re: Bone Growth

 

 

 

Chinese Traditional Medicine@ <Chinese Traditional Medicine%40>

, " Shoshanah " <ositaguapa

wrote:

> The kidneys rule the bones, and since I had

> been having kidney problems for years, when that was going better,

> bone growth would follow.

 

I want to emphasize this for the beginning TCM students on the list.

Any time there are bone problems suspect and rule in or rule out

possible Kidney imbalance. The health of the Kidneys often are

reflected in the bones and the teeth.

 

I also want to emphasize that you'll run across cases where the

kidneys are OK according to allopathic standards, but the Kidneys are

severely imbalanced.

 

Bone problems are a general indicator of possible Kidney imbalance.

You'll need to pay attention to other signs and symptoms to narrow

the diagnosis down to which particular Kidney imbalance (if there is

one). Kidney Yang Deficiency, Kidney Yin Deficiency, Kidney Qi

Deficiency, Kidney Jing Deficiency, etc.

 

 

 

 

 

 

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