mad cow dz and gu syndrome

[Guest guest]

According to Heiner Fruehauf (JCM 57 MAY 1998), there are five primary

etiological factors in gu transmission. Let's see how they mesh with

our knowledge of mad cow dz.

 

1. gu is transmitted by food (in the case of MCD, it is the eating of

rendered infected sheep brain in animal feed given to cows, which is

somewhat an abomination of nature, as cows are very strict vegetarians).

 

2. gu may be life threatening (MCD is always fatal)

 

3. gu represents a type of toxin (gu du - many toxins as described in

TCM have viral asociations, as does MCD)

 

4. gu thrives in already vacuous organisms and causes further vacuity

(MCD only occurs in specific genetic cluster, which may imply an

inherited weakness in resistance, which may appear as zheng qi xu)

 

5. gu operates in the dark. it is often unclear when and how the

pathogen was contracted (MCD infection occurs without symptoms from

eating an apparently harmless food; there is a long latency period

before an apparently healthy person is stricken)

 

The mains sx of gu are severe mental and GI. MCD is characterized by

severe progressive dementia and derangements of every aspect of nervous

system function. While GI sx are not keynotes of this clinical picture,

it is easy to see how a wide variety of GI symptoms could result from an

infection affecting the sympathetic and parasympathetic nerve plexi.

 

[Guest guest]

on mad cow...something i read about it the other day...although my knowledge

of physiology is pitiful...

 

mad cow can arise not only from eating contaminated food, but endogenously.

after a high or prolonged fever albumin levels may drop. Albumin seems to

prevent the degenerative process causing MC, which is protein segments

becoming doubled over and forming into prions, this process then being

repeated by contact with other protein segments in a chain reaction.

 

albumin drop after a fever - what's this in chinese terms? latent heat in

the blood?

 

Many older people suffering dementia may have MC rather than Alzheimers.

 

deficient Ki Yin?

 

 

Andrew

 

- <herb-t

cha

Friday, 28 July 2000 7:29

mad cow dz and gu syndrome

 

 

> According to Heiner Fruehauf (JCM 57 MAY 1998), there are five primary

> etiological factors in gu transmission. Let's see how they mesh with

> our knowledge of mad cow dz.

>

> 1. gu is transmitted by food (in the case of MCD, it is the eating of

> rendered infected sheep brain in animal feed given to cows, which is

> somewhat an abomination of nature, as cows are very strict vegetarians).

>

> 2. gu may be life threatening (MCD is always fatal)

>

> 3. gu represents a type of toxin (gu du - many toxins as described in

> TCM have viral asociations, as does MCD)

>

> 4. gu thrives in already vacuous organisms and causes further vacuity

> (MCD only occurs in specific genetic cluster, which may imply an

> inherited weakness in resistance, which may appear as zheng qi xu)

>

> 5. gu operates in the dark. it is often unclear when and how the

> pathogen was contracted (MCD infection occurs without symptoms from

> eating an apparently harmless food; there is a long latency period

> before an apparently healthy person is stricken)

>

> The mains sx of gu are severe mental and GI. MCD is characterized by

> severe progressive dementia and derangements of every aspect of nervous

> system function. While GI sx are not keynotes of this clinical picture,

> it is easy to see how a wide variety of GI symptoms could result from an

> infection affecting the sympathetic and parasympathetic nerve plexi.

>

 

>

>

>

>

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