Guest guest Posted August 11, 2004 Report Share Posted August 11, 2004 Here is some additional information I thought I would pass your way. Brian N Hardy, DC, LAc, CCN EVALUATING BLOOD TESTS FROM A TCM PERSPECTIVE The following is a list of common serological tests. The General Laboratory Values are first listed. The ptimum values are what is preferred. In the left hand column you will find the Western medical problem; in the right hand column you will find the common syndrome most often found in combination with the medical problem and the elevation or depression of the blood chemistry. Key: ^ & > = increased; more, heightened; intensified < = decreased; less, diminished BLOOD RED BLOOD COUNT (RBC) The red blood cell is a carrier of oxygen through the hemoglobin it contains. The RBC value measures the actual oxygen carrying ability of the blood. General Laboratory Values: Male: 4.4-6.2 million cu.MM Female: 3.8-5.4 million cu.MM Red Blood Cell Increased In: B6 Anemia Sp, Kid Qi Xu Emphysema (check HGB) Sp, Kid, Lu Zi Xu; Lu Yin Xu Respiratory Distress (check total protein) Kid Qi Xu; Lu Qi Xu Adrenal Hyperfunction (check potassium) Kid Damp Heat Cystic Fibrosis Sp Qi Xu; Liv Stagnation Damp Heat Red Blood Cell Decreased in: Iron Anemia (check HGB) Sp Qi Xu; Lu Qi Xu Folic Acid Anemia (check MCV/MCH) Sp Qi Xu Hereditary Anemia <Jing Qi; Sp Qi Xu Liver Dysfunction (check SGPT) Li Stagnation; Li Heat Renal Dysfunction (check creatinine) Kid Qi Xu Free Radical Pathology <Wei Qi; Sp, Kid Qi Xu General Picture of Toxic Metal Poisoning Increased Eosinophils, Bilirubin, BUN Decreased RBC, HCT, HGB, MCV, MCH General Picture of Cadmium Poisoning (including toxic metal) Increased SGOT, SGPT, Calcium Decreased Phosphorous General Picture of Mercury Poisoning (including toxic metal) IncreasedSGOT, SGPT Decreased CO 2 General Picture of Arsenic Poisoning (including toxic metal) Increased Alkaline Phosphatase RX: Toxic Metal Poisoning Methionine, L-Cysteine, L-Glutathione sulfur amino acids detoxify metals Selenium/Vitamin E anti-oxidant Zinc - <zinc associated with toxic metal > Calcium/Magnesium prevents lead particularly from depositing in body (^EOS) Vitamin C heat in bld; (^EOS; ^Uric Acid) Garlic helps to bind and excrete toxic metals esp. lead Milk Thistle Extract hepatoprotective; (^EOS; ^BUN; ^Uric Acid) Isatis Formula w/ infx; toxic heat in liver; (^BUN; ^Uric Acid) Fiber Combination removes toxic metabolites more quickly; broom effect Siberian Ginseng Combination adaptogenic; reduces metal and radiation poisoning NOTE: After detoxification is achieved use an adaptogen such as American Ginseng, Siberian Ginseng, or Siberian Ginseng Combination one month per year is an excellent prophylactic measure. HEMATOCRIT (HTC) Hematocrit represents the packed cell volume of red blood cells. It is the percentage of the total volume occupied by packed red blood cells when a given volume of whole blood is centrifuged at a constant speed for a constant period of time. HCT is one of the most precise ways of measuring the degree of anemia. Hematocrit combined with serum iron and hemoglobin is a diagnostic tool for determining iron excess or deficiency. General Laboratory Values: Males: 39-54% Females: 35-48% Hematocrit Increased In: Dehydration Kid Yin Xu; Lu Yin Xu; Ht Bld Xu Asthma Spleen Qi Xu; Kid Qi Xu; Lu Qi Xu Emphysema Spleen Qi Xu; Kid Qi Xu; Lu Qi Xu; Lu Yin Xu Respiratory Distress Kid. Qi Xu; Lu Qi Xu Adrenal Hyperfunction Kidney Damp Heat Diarrhea Spleen Damp Heat; LI Damp Heat Rx. Hematocrit Increased Capilaris & Hoelen Formula Damp Heat (diarrhea; adrenal^) Hoelen 5 Formula - Dampness (diarrhea; adrenal hyperfx) Hematocrit Decreased In: Parasites LI Stagnation; Liver Stagnation, Sp Qi Xu Adrenal Hypofunction Kid Qi Xu Anemias Spleen Qi Xu; Lu Zi Xu; LI Xu; Liver Bld. Def. Digestive Inflammation Spleen Qi Xu; Damp Heat Spleen Liver Dysfunction Liver Stagnation; Live Heat Renal Dysfunction Kid Qi Xu Rheumatoid Arthritis Spleen Qi Xu; Kid Qi Zu, <Wei Qi; Liv. Stag. Rx. Hematocrit Decreased Ginseng & Longan Formula - Qi/Blood Def (B 12 def; folic acid def; fe<) Ginseng Nutritive Formula - Qi/Blood Def (B1 def; B12<; fe<) Tang Kuei & Ginseng 8 Formula - Qi/Bld Def (B-12<; adrenal<) Minor Bupleurum Formula - Liv Stag.; St Phlegm; (Liv dysfx; parasites) Bupleurum & Cinnamon Formula - Liv Stag. w/Ht; (Liv dysfx; rheumatoid arthritis) HEMOGLOBIN (HGB) Hemoglobin is the circulating iron containing pigment, which carries oxygen from the lungs to the tissues. It is a measurement of how earth feeds metal. Hemoglobin ability to transport oxygen depends upon pH and the presence of ferrous iron. Hemoglobin is the most abundant protein found within the red blood cell. Hemoglobin level measures the amount of intracellular iron. Hemoglobin is synthesized in most bodily tissues but the liver is the largest heme producing organ. (The muscles being fed by iron as well as glucose liver). In the bone marrow heme is transformed into hemoglobin. It is also a measurement of how the metal manipulates the wood. It is important to note that infants have a higher hemoglobin level than adults. (growth/wood excess in infants). Hemoglobin is considered along with hematocrit, red blood cells, MCV and MCH in determining anemia. Ideally serum iron and ferritin will also be measured. General Laboratory Values: Males: 13.0 18.0 G/DL Females 11.5 16.0 G/DL Hemoglobin Increased In: Asthma Spleen Qi Xu; Kid Qi Xu; Lu Qi Xu Emphysema Spleen Qi Xu; Kid Qi Xu; Lu Qi/Yin XuAdrenal Dysfunction Kidney Damp Heat Diarrhea/Dehydration Spleen Damp Heat; LI Damp Heat Serum Profile of Asthma Increased HGB, HCT QBC, Eosinophils Decreased Lymph, Potassium Rx: Asthma Homeopathic Remedy - Arsenicum Album; Pulsatilla; Phosphorous Antronex - Liver Stagnation Pancreatic Enzyme - Sp Qi Xu Adrenal Extract - Kid Qi Xu Calcium/Magnesium - will alleviate wiry pulse Minor Blue Dragon Formula - Phlegm/Damp; Lu Qi Zu; ^EOS Pinellia X Formula - Phlegm/Damp Heat; sinus; ^LDH, ^EOS, <K Pueraria Formula - Wind Cold; GB congestion; ^EOS, ^WBC Hemoglobin Decreased In: Parasites LI Stagnation; Li Stagnation; Sp Qi Xu Adrenal Hypofunction Kid Qi Xu Anemias <Wei Qi; Lu Qi Xu; Sp Qi Xu; Kid Xu; Jing Qi Xu Digestive Inflammation Sp Qi Xu; Sp Damp Heat Liver Dysfunction Li Stagnation; Li Heat; Li Heat Rising Renal Dysfunction Kid Qi Xu] Rheumatoid Arthritis Spleen Qi Xu; Kid Qi Xu; <Wei Qi Serum Profile of Iron Anemia Increased B 12, Globulin above 2.8 Decreased RBC, HCT, HGB, Iron, Bilirubin MCV, MCH, MCHC Rx: Iron Anemia Alfalfa Extract - TID two weeks; BID two weeks Liver Extract - TID (<HCT, <HGB, <Fe, <Bilirubin) HCL - with meals (<Fe) Floradix - TID (<HCT, <HGB) Dong Quai Combination - Blood Def; w/Sp Qi Xu) Tang Kuei Four Formula - Blood Def; anemia<aft. Mense; (<HCT, HGB, Fe, RBC) Serum Profile of B12 Anemia Increased HGB, MCV, MCH, Iron Decreased WBC, Cholesterol Eosinophils Potassium, B12, Albumin Rx: B12 Anemia Folic Acid/B12 400 mcg/500 mcg TID 2 weeks 400 mcg/500 mcg BID 2 months Liquid B Complex one dose BID 2 months ]Thymus Extract two tablets TID 2 weeks Tang Kuei & Ginseng Form. Sp & Liv Bld Def; fatigue (MCV^; MCH^, WBC<) following mense or illness Ginseng & Longan Formula Sp Qi Xu; calmative; insomnia Ginseng Nutritive Formula Geriatric anemia (^MCV; ^MCH; <WBC) Floradix Consider for vegetarians and Cold Sp/St Dong Quai Combination Sp Qi Xu; thyroid imbalance Serum Profile of Folic Acid Anemia Increased MCV; MCH; Iron; LDH Decreased RBC; HGB; HCT; Folate, Uric Acid, Potassium Rx: Folic Acid Anemia Folic Acid/B12 400 mcg/500 mcg TID 2 weeks 400 mcg/500 mcg BID 2 months Liquid B Complex one dose BID 2 months Alfalfa Extract dose TID / Tang Kuei & Peony Formula Bld Xu; Kid Qi Xu; (MCV>, Folate<; HGB<; HTC<) Ginseng & Astragalus Formula Sp Qi Xu; (Folate<, RBC<, MCV>) Notations It is difficult to differentiate between folic acid anemia and B-12 anemia. One generalization: if the patient is suffering with a looser stool, supplement first with folic acid. If the patient has leanings more toward constipation, use B-12 first. With both anemias the Qi of the Stomach is diminished. Herbal Spleen Qi enhancing formulas such as Ginseng and Astragalus as well as/or HCL should be considered. B-12 and Folic Acid should be considered when dealing with inflamed nerve tissue or degeneration (myalgias), blood sugar disorders, vegetarians and pregnant women. (Pregnancy/neural tube defect). Iron anemia could be the outcome of a history of excessive aspirin use. If sclera of the eyes are blue, need for iron. MEAN CORPUSCULAR VOLUME (MCV) MCV indicates the volume in cubic microns occupied by an average single red blood cell. MCV increases or decreases with an increase or decrease in MCH is a finding for folic acid/B-12 deficiency (increase) or iron, copper or B-6 (decrease). General Laboratory Values: 80 100 cuMM MCV Increased Folic acid/B-12 anemia Hereditary anemia Decreased Parasites (check eosinophils) Iron anemia (check HGB) Hypochlorhydria (check food allergies) B-6 anemia; Vit. C anemia Rheumatoid arthritis; Lead poisoning MEAN CORPUSCULAR HEMOGLOBIN (MCH) MCH indicates the weight of hemoglobin in a single red blood cell. When MCH >s or <s with an > or < in MCV it is an indicator for folic acid and/or B-12 deficiency. A < in MCH with a < in MCV will point to an iron, B-6 or copper deficiency. General Laboratory Values: 27 33 micro micrograms MCH Increased B-12/Folic Acid anemia Hereditary anemia Decreased Parasites Iron anemia, Copper deficiency Rheumatoid Arthritis (check Alk Phos) Toxic metal poisoning lead, etc. MEAN CORPUSCULAR HEMOGLOBIN CONCENTRATE Indicates the hemoglobin concentration per 100ml of packed red blood cells. General Laboratory values: 31 36% PLATELETS Platelets, or thrombocytes, are tiny bits of cytoplasm, much smaller than the red blood cells but lacking a nucleus. They are round or biconcave disks and are normally about 30 to 40 times more numerous than the white blood cells. They are produced as broken fragments of the cytoplasm of the giant cells of the bone marrow the megakaryocytes. The platelets primary function is to stop bleeding, with the help of certain clotting proteins in the plasma. When tissue is damaged, the platelets aggregate in clumps to obstruct blood flow through the smallest vessels, the capillaries. In the larger vessels, the clumps of platelets form a site around which a blood clot forms, aided by a clot-promoting factor freed by the platelets as they break down. In short, platelets are responsible in the blood clotting mechanism. General Laboratory Values: 150,00 450,00 CuMM Platelets Increased Anemia / Rheumatoid Arthritis Decreased Anemia / Liver Dysfunction; Lupus QI WHITE BLOOD CELLS (TOTAL WHITE BLOOD CELL COUNT) White Blood Cells are divided into two groups: 1) Granulocytes: neutrophils, eosinophils and basophils, 2) Nongranulocytes: lymphocytes and monocytes. General Laboratory Values: 4 11 Thous./cu.mm. WBC Increased Measles, Mumps (onset) Mono (2nd/3rd wk) Parasites Adrenal Dysfunction Infection (abscess) Flu w/additional infx. Asthma Decreased Measles, Mumps (Late) Mono (1st wk/chronic) Parasites Adrenal Dysfunction Chronic Infection (viral/bacterial) Flu (onset) / B-12, B-6, Folic Acid Anemia / Lupus Erythematosis (LDH^; SED^) / Monos^; Cholest^; Albumin<; HCT/HGB<; Lymphs<; Platelets< RX: Elevated WBC Week #1 Yin Qiao one dose QID first 48 hours Echinacea Combination one dose TID/QID Vitamin C mineral ascorbate 500-1000 MG QID Thymus Extract 2 tablets TID Proteolytic Enzymes dose TID betw. Meals Consider stomach protective measurement Week #2 Isatis Formula dose TID Zinc Picolinate 15 mg. BID Consider short course of pancreatic enzyme for Spleen Deficiency Week #3 Minor Bupleurum Formula dose TID Thymus Extract dose TID NEUTROPHILS Neutrophils are predominantly involved with phagocytosis. Neutrophil count is a way of determining the strength of the immune system ability to fight infection. General Laboratory Values: 45 75 percent of total WBC NEUTROPHILS Increased Infection Adrenal Dysfunction; Gout Pregnancy Last Trimester Decreased Infection Bone Marrow Depression Iron, B-12, Folic Acid BANDS OR NON-SEGMENTED NEUTROPHILS Bands are the youngest form of neutrophils typically found in the peripheral blood. They increase during acute infection with or without an increase in the total WBC. BAND measurement is useful in determining an infectious process. General Laboratory Values: 0 10% of total WBC BANDS Increased Acute Infection Decreased Not considered a significant finding LYMPHOCYTES Lymphocytes help to destroy toxic metabolites of protein metabolism. They originate from lymphoblasts in the spleen, lymph, glands, tonsils, bone marrow and thymus. They help to determine the stage of infection: acute; recovery; chronic. Generally when lymphs are high there is some systemic toxicity; when lymphs are low there is often chronic viral infection. General Laboratory Values: 18 46 percent LYMPHOCYTES Increased Infection; Flu Parasites Anterior Pituitary Hypofunction Adrenal Dysfunction Hyperthyroidism Decreased Infection; Flu Lupus Late Pregnancy Adrenal Dysfunction Immune Deficiency RX: Acutely Increased Lymphocytes Day #1: Aconite 200x one dose Yin Qiao one dose QID Curing Formula one dose TID Days #2 5: Echinacea Combination one dose QID Consider additional Echinacea Vitamin C 1,000 MG QID Wobenzyme one/two with meal Consider proteolytic enzymes between meals Days #6 10 Isatis Formula one dose QID Acidophilus/Bifidus one dose BID Wobenzyme one/two with meal Zymex II 3 caps two times daily between meals Jade Screen Formula dose BID MONOCYTES The monocyte is a young macrophage. The cells that line sinusoids in spleen, liver and lymph notes derive from the same monocyte-macrophage pool. Normal macrophages have enzyme systems capable of synthesizing and of degrading sphingolipids, compounds important in biologic membranes and especially prominent in the nervous system. Monocytes are of primary importance in diagnosing mononucleosis. At the onset of illness the WBC may be low but by the end of the first week the count will usually be between 10,000 - 30,000mm. There is an increase in lymphocytes as well as monocytes which will often be over 15%. General Laboratory Values: 0 - 10% MONOCYTESIncreased Parasites Hepatitis (SGPT) Benign Prostate Hypertrophy (Creatinine) Acute Infection; Mumps DecreasedNot Significant RX: ELEVATED MONOCYTES Addressing liver stagnation is most important. Simultaneously consider blood cleansers, anti-virals and liver regulators. EXAMPLE Day #1: Echinacea Combination - 2 droppers 5x daily Vitamin C - 1,000 mg. - 5x daily Appropriate Homeopathic - 4x daily (Oscillococinnum) Days #2, 3, 4: Echinacea Combination - 2 droppers 3x daily Isatis Formula (with stronger spleen) - 2 droppers 3x daily Adrenal Extract and Vitamin C - QID Days #5,6,7: Isatis Formula - 2 droppers 2x daily Minor Bupleurum Formula - 2 droppers 3x daily Merc. Sol, Merc. Viv, or Merc. Dulc. - dose 3x daily EOSINOPHILS (EOS) Eosinophils are the mediators in acute inflammation and increase with allergies, some skin disease, after radiation exposure and parasites. EOS are involved with the detoxification and removal of excess proteins. W/ elevation food sensitivities are important to consider. Elevated eosinophils are decreased with adrenal support. It is important when regulating EOS that urinary and saliva pH is maintained between 6.0-6.5. General Laboratory Values: 0-6% EOSINOPHILSIncreased Decreased Allergies (IgE) not significant Asthma (IgE/HGB) Rheumatoid Arthritis (Alk. Phos.) Hayfever; Dermatological Disorders Parasites (IgE/<Fe/MCH/MCV/HGB/HCT) Hyperthyroidism (T3 Uptake) Adrenal Cortical Hypofunction (K) / Anterior Pituitary Hypofunction (TSH) RX: ELEVATED EOSINOPHILS Week #1: Rule out need for HCL Echinacea Extract - 2 droppers TID Pancreatic Enzymes - dose w/ meals Zymex - (for children) - dose BID Thymus Extract - dose TID Week #2: Pancreatic Enzymes Adrenal Extract - dose TID Vitamin C - dose TID Symptom specific formula: Minor Bupleurum; Minor Blue Dragon; Du Huo and Loranthus; Pueraria comb.; or Milk Thistle Extract Calcium/Magnesium - dose BID Week #3: Syndrome specific herbal formula Thymus/Adrenal as needed Vitamin C - BID Zinc - BID B-12 - BID BASOPHILS (BASO) Basophils contain enzymes called lysosomes. Lysosomes activate the release of histamine and hyaluronic acid. Basophils release heparin essential to fighting inflammation and preventing clotting of the blood in inflamed tissue. Hyalouronic acid is an interstitial adhesive/protective factor whose production is associated with cortisol output. General Laboratory Values: 0 -2% BASOPHILSIncreased Parasites (EOS) Chickenpox (WBC) Flu; Inflammation Hypothyroidism (T3/T4) Chronic Hemolytic Anemia (RBC/HGB) Decreased not significant FIRE: HEART/PERICARDIUM/SMALL INTESTINE SEDIMENTATION RATE General Laboratory Values: Male - 0 - 15 MM/HR / Female - 0-20 MM/HR The Sedimentation Rate helps to determine inflammation and/or destruction within a disease process. It helps in following the course of an established condition and also signals the onset of inflammation. Fibrogen increases the Sed. Rate, while an increase in albumin decreases the Sed. Rate. The liver is the seat of albumin synthesis. A damaged liver, therefore, can contribute to low albumin with a corresponding increased Sed. Rate. SEDIMENTATION RATE Increased Inflammation Decreased Not significant THYROID Thyroid Hormones Control of oxygen consumption is the most conspicuous biologic effect of the thyroid hormones, a physiologic variable measured in simplest fashion by the basal metabolic rate. Thyroid hormones also influence carbohydrate and protein metabolism, and the mobilization of electrolytes, and the conversion of carotene to vitamin A. (wood/fire). Although the mechanism is not fully apparent, thyroid hormones are essential for development of the CNS and the thyroid deficient infant suffers irreversible mental damage. The thyroid deficient adult may have slowed deep tendon reflexes. Thyroid hormones affect synthesis and metabolism of fats. Abnormalities within the endocrine system may be reflected in altered lipid levels. In hyperthyroidism, degradation and excretion increase more than synthesis, resulting in low levels of cholesterol and triglycerides. Hypothyroidism slows catabolism more than it affects synthesis, and hypercholesterolemia and hypertriglyceridemia. Hypothyroidism secondary to pituitary failure, however does not cause lipids to rise. In an obviously hyypothyroid patient, a normal serum cholesterol level should direct attention to the pituitary. (Is the problem wood or water). Cholesterol levels will often drop within 3 weeks after thyroid medication. The thyroid gland synthesizes its hormones from iodine and the essential amino acid tyrosine. Most of the body's iodine enters through the alimentary tract as iodide, but under certain circumstances, the lungs and skin may be portals of entry. Of the iodine that enters the body, approximately one third enters the thyroid gland and two thirds leaves the body in urine. Enzymes oxidize iodide to organic iodine, which is incorporated into monoiodotyrosine and diiodotyrosine. These one and two iodine containing compounds are building blocks for the active thyroid hormones T4 which has four iodine molecules, and triiodothyronine T3, which has three. T-3 UPTAKE Useful to dx. Hyperthyroidism. General Laboratory Values: .8 - 1.2 T-3 Increased Hyperthyroidism Protein Malnutrition Renal Dysfunction Decreased Hypothyroidism; Pregnancy Estrogens/Anti-Ovulatory Drugs Triiodothyronine Rx. for Hypothyroidism Propylthioouracil Rx. for Hyperthyroidism T-4 THYROXINE General Laboratory Values: 4.5 - 12 mcg/100m. T-4 is a product of the thyroid follicular cell. It influences the entire body's metabolism. In primary hypothyroidism T-4 levels are usually low. In many cases of sub-clinical hypothyroidism (chk. Daily temperature), T-4 levels may be low normal. T-4 Increased Hyperthyroidism Liver Cirrhosis Pregnancy Decreased Hypothyroidism, Renal Dysfunction Diabetes; Anterior Pituitary Dysfunction Protein Malnutrition THYROID STIMULATING HORMONE (TSH) TSH is secreted from the pituitary gland. It regulates the uptake of iodine as well as the synthesis and secretion of the thyroid hormones. TSH is influenced by hypothalamic stimulation as well as T4 concentration. General Laboratory Values: .4 - 6 mcIU/ml TSH Increased Hypothyroidism Liver Cirrhosis Decreased Hyperthyroidism Anterior Pituitary Hypofunction Hyperthyroidism Increased T4, T3 Uptake; Glucose; BUN Alkaline Phos; Eosinophils Decreased Basophils; Albumin; Calcium Total Protein; HCT; HGB; Iron Magnesium; Cholesterol; Triglycerides Hypothyroidism Increased LDH, Calcium, Magnesium Basophils; Cholesterol; Triglycerides Decreased T3 Uptake; T4; HCT; HGB; Iron Sodium SYMPTOMATOLOGY Hypothyroidism >Cholesterol, Triglycerides >Serum Carotene (yellow skin) >Muscle Enzymes: CPK, AST, LDH >Prolactin Anemia; HGB @ 10g/dl Capillary fragility Spinal fluid protein Hyperthyroidism <cholesterol, triglycerides >skin temp, pulse rat, pulse pressure >Alkaline Phosphatase Altered glucose/insulin relationship >Proportion of lymphs in differential WBC >Urinary calcium excretion Pituitary/Hypothalmus The thyroid produces hormones upon stimulation by the pituitary hormone variously called thyrotropin or thyroid stimulating hormone (TSH). Pituitary production of TSH (Kidney energy) follows stimulation by a hypothalamic protein called thyrotropin releasing hormone (TRH), which responds to activity levels of T3 and T4 in the blood passing through the hypothalamus. When hormone levels are low TRH provokes TSH secretion, which then accelerates all aspects of thyroidal iodine metabolism and hormone production. EARTH: STOMACH/SPLEEN GLUCOSE This test is a useful indicator of glucose metabolism disorders. Glucose represents the synthesis of carbohydrates and is the form in which carbohydrate is supplied to the cell from body fluids. In other words, glucose is the essence (yin) of carbohydrate metabolism. Insulin, of particular importance in the transport of glucose into the fat cell, lowers serum glucose while adrenal and pituitary hormones tend to elevate it through their effects on the liver. The thyroid gland, on the other hand, by helping with the selective activity of the intestinal tract for sugar, increases blood sugar if it is overactive and decreases blood sugar if it is under-active. Since brain cells cannot derive energy from anaerobic metabolism of glucose, they are most vulnerable to hypoglycemia (enter brain fog General Laboratory Values: 65-115 MG/DL Glucose Increased Diabetes Damp Heat in Spleen; Liv. Stag. Acute/Chronic Pancreatitis Damp Heat in Spleen Digestive Inflammation Damp Heat in Spleen Digestive Inflammation Damp heat in Spleen Hyperthyroidism Damp Heat in Spleen; Liv. Stag. Pregnancy Damp Heat in Spleen Adrenal Cortical Hyperfunction Damp Heat in Kidney Chronic Renal Dysfunction Damp Heat in Kidney Cardiac Dysfunction Damp Heat in Heart Glucose Decreased Hypothyroidism Spleen Qi Xu Malnutrition Spleen Qi Xu Pregnancy Spleen Qi Xu Hypochlorhydria Spleen Qi Xu Liver Dysfunction Spleen Qi Xu; Wind Attacking Earth Hypoglycemia Spleen Qi Xu; Kidney Qi Xu Anterior Pituitary Dysfunction Kidney Qi Xu Adrenal Cortical Hypofunction Kidney Qi Xu Diabetic Blood ProfileIncreased Glucose; Basophiles; Creatinine GGTP, Triglycerides; Cholesterol BUN; Uric Acid; Alk. Phos & LDH normal -^ Decreased Calcium; Insulin; Lymphocytes; T-cells Blood pH; Phosphorous; Chloride Albumin & Magnesium normal to < It is interesting to note that with diabetes or pre-diabetic condition, the serum triglycerides will often be higher than the serum cholesterol. In Oriental Medicine this would relate to the predominant problem being Damp Heat in the Spleen as opposed to ure Liver Stagnation. RX: HYPERGLYCEMIA (HERBAL) Astragalus & Ganoderma Formula Deficiency of Qi and Yin Anem-Phello & Rhemannia Formula Heat w/ Yin Deficiency Bitter Herb Combination Liv Stag.; Phlegm in Stomach/Sp. Bupleurum/Dragon Bone Formula Diabetes w/ hypertension Dandelion Extract Liver Stag.; ^Cholesterol/Trigs. Goldenseal Extract Heat in St.; Dampness; infx. Echinacea Combination if associated with infection Garlic Hyperglycemia w/atherosclerosis RX: HYPERGLYCEMIA (NUTRITIONAL) Chromium 100 mcg. BID Stabilizes blood sugar L-glutamine 500 mg. BID between meals or beg. meal; sustains blood sugar Niacin/Niacinamide 100 mg. 2X daily every other week 3 months Lipotropics (Choline, Methionine, Inositol) 350 mg. BID digest fats Zinc (taste test) 15 mg. BID; slow healing; check nails; fungal infx. Vit. A (emulsion or cap) 15,000 IU diabetics have difficulty converting beta-carotene to Vit. A Vit. C Mineral ascorbate 1,000 mg. TID Vit. E Caution with hypertensive diabetic 200 IU BID TID TEST WHICH CAN BE DONE IN THE CLINIC Those who have an onset of diabetes with adulthood (type II diabetics) are not able to perceive sweet tastes easily. The taste buds must be very stimulated with a large amount of concentrated sweet for the taste to be perceive. It almost as if the Spleen has lost some of its intelligence, discerning power or Qi. This patients inability to perceive sweet tastes makes it very difficult for them to lose weight. WHY? They do not recognize sweet foods as such, and will often consume large amounts of sugary type foods. This test will often detect an impaired ability to taste sweets. STEP #1: Make sure the patient has not consumed caffeinated stimulants within the last three hours. STEP #2: Label seven glasses as: 1: No sugar; 2: tsp. sugar; 3: tsp. sugar; 4: 1 tsp. sugar, 5: 1 tsp. sugar; 6: 2 tsp. sugar; 7: 3 tsp. sugar. Place these amounts of sugar in eight ounces of water. STEP #3: Have the patient sip with a straw from each glass. After each taste have them rinse their mouth with water. Most people without a sugar disorder will be able to perceive a sweet taste with only one teaspoon or less of sugar in the eight ounces of water. Most individuals with a sugar disorder will need 2 to 3 teaspoons of sugar present in the eight ounces to perceive sweetness. THE HYPOGLYCEMIC BLOOD PROFILEIncreased Growth Hormone^ Insulin^ Decreased Glucose < LDH normal to < A hypoglycemic may display any or all of the following symptoms: fatigue, dizziness, headache, irritability, depression, anxiety, tightness in the chest, sweet cravings, confusion, night sweats, weakness in the legs, nervous habits, insomnia and an assortment of pains. These symptoms relate to various syndromes, the most obvious being Spleen Qi Xu, Kid. Qi Xu and Liver Stag. NOTATIONS: A low normal glucose combined with a low normal LDH is reason to suspect hypoglycemia. In Oriental Medicine this would be associated with Spleen Qi Xu. Hypochlorhydria (St. Def.) and biliary stasis (Liver Stagnation) are prominent syndromes in most cases of hypoglycemia. Low blood pressure (Kid. Qi Xu) is also common amongst hypoglycemics. With blood sugar problems, a craving for sweets is common. Bile salts for a short period of time 2 weeks, along with chromium, is an effective way to diminish this sweet craving. Ginseng & Astragalus and/or Ginseng & Atractylodes Formulas will enhance digestion and help to take the extreme dip out of the blood sugar curve. Homeopathics: Pulsatilla; Arg. Nit. Headaches associated with chronic low glucose: Ginseng & Astragalus Formula Hypoglycemia prior to menstruation is often due to a poor diet of excess sugar and fats. Harmonizing the liver/gallbladder with Bupleurum and Tang Kuei Formula (for the more Blood Def.) of Bupleurum and Paeonia Formula (for the more Liver Stag. wiry pulse) will assist in controlling the blood sugar before menses. A flat glucose curve is common with learning disabilities and ersonality disorders or disturbed Shen. Heavy metals may also be the culprit here. Food allergies, especially gluten and milk, may often cause blood sugar fluctuations. If LDH and glucose are low and such symptoms as hypochlorhydria, (Sp. Qi Xu) hypotension and adrenal fatigue, (Kid. Qi Xu); and acidic urine and saliva (Stagnation) one must rule our food allergies. Avoidance of allergens, simple sugars and over-cooked fats is a must. RX: HYPOGLYCEMIA (HERBAL) Ginseng & Astragalus Formula Sp. Qi Xu; prolapse; taken between. Meals will help to control blood sugar; taken w/ meals assists digestion. Saussurea & Cardamon Formula Sp. Qi Xu; Damp; St. Stagnation; gas; w/meals to resolve digestive symptoms Ginger Extract Cold; Stagnant St.; w/ meals for digestion; between meals for nausea, morning sickness Echinacea Extract low grade infx. (5 days 2x each month 3 months) Licorice Extract may be added to Ginseng and Astragalus Formula. By itself effective as a Kid. Qi tonic for one week at a time. Administer at 11 AM and 3 PM. (do not use with hypertensives or individuals with sodium retention. Used historically in the treatment of Addison Dx (exaggerated Kid. Qi Xu). Licorice Extract enhances endogenous activity of mineralocorticoids via inhibition of degradation by the liver. Glycyrrhizin suppresses 5-beta-reductase, the main enzyme responsible for inactivating cortisol, aldosterone and progesterone. Glycyrrhiza also has significant anti-inflammatory and anti-allergic activity. Glycyrrhetinic acid was the first drug shown to treat peptic ulcers. Glycyrrhetinic acid also effective in speeding the healing of oral and genital herpes. RX: HYPOGLYCEMIA (NUTRITIONAL) Chromium Bile Salts Adrenal Extract Liquid B Complex p 50 mg. 2x daily imperative in carbohydrate metabolism. Augments Spleen Qi. Will create heat and is therefore not as effective in diabetes as in hypoglycemia. B-12 500 mcg. As needed (may be given IM 1 cc. Every 5 days for 4 doses. Will assist in bowel absorption of nutrients. Enhances communication or feeding from the earth to metal. Assists in resolution of diarrhea/constipation associated with food allergies/hypoglycemic symptoms. Note: a useful injection: B complex (1 cc) plus pyridoxine (1/2 cc) once per week for one month has shown to be very effective in the beginning stages of treatment for hypoglycemia. This is a relatively mild treatment and can be combined with any of the herbal formulas. ACE injections in small amounts will also assist in initial stages of tx. Pantothenic Acid 350 mg. BID/TID magnesium is important in sugar metabolism. Lipotrophics (Choline, Methionine, Inositol) Liv. Stag.; ^lipids Fiber Combination (Guar Gum, Psyllium, etc.) LI Stag.; may create a problem w/ Sp Qi Xu. Begin with small amounts. METAL: LUNG/LARGE INTESTINE CARBON DIOXIDE (CO2) General Laboratory Values: 22-32 mEq/L In the combustion of food, oxygen is used and carbon dioxide is given off. The rate of oxygen consumption indicates the energy expenditure of an organism, or its metabolic rate. The metabolic rate of any given animal at any given time is highly variable and is influenced by many diverse factors, including amount of muscular activity; quality of diet; presence or absence of digestion, lactation or pregnancy; time of day or year; period of the menstrual cycle and emotional state. Although most CO@ is lost through the lungs, some is converted to bicarbonate. This bicarbonate is part of the alkaline reserve available for neutralization of acids. Arterial blood has a lower total CO2 than venous blood. CO2 Increased Fever; Respiratory Distress/Alkalosis Adrenal Hyperfunction Decreased Dehydration; Acidosis Renal Dysfunction TOTAL IRON Iron is largely absorbed across the mucosa of the duodenum and proximal jejunum. Gastric juice plays an important but not thoroughly understood role in promoting absorption. The low pH of gastric juice makes iron more available from iron-rich foods. General Laboratory Values: 40 170 mcg/mL IRON Increased Pernicious Anemia/B-12 Liver/Kidney Dysfunction Decreased Iron Anemia; Parasitic Infection Liver/Kidney Dysfunction; Gastrointestinal Dx. (peptic ulcer; diverticulitis) CALCIUM General Laboratory Values: 8.5 10.5 mg/dl Calcium is absorbed from the upper part of the small intestine. Absorption depends upon the relative acidity of the intestinal contents and the amount of phosphate present. Calcium absorption is influenced by parathyroid hormone, calcitonin and Vitamin D. Calcium Increased Hyperparathyroidism Ovarian Hyperfunction Epilepsy Decreased Hypoparathyroidism Osteoporosis; Acidosis; Hypochlorydria Ovarian Hypofunction PHOSPHOROUS Phosphorous plays an important role in the hemostasis of calcium and in reactions involving carbohydrates, lipids, and proteins. The chemical energy of the body is stored in igh energy phosphate: compounds. Calcium and phosphorous dynamics are largely regulated by the effects of parathyroid hormone on bone and on urinary excretion and the effects of vitamin D metabolites on intestinal absorption and on bone. Parathyroid hormone causes increased resorption of both calcium and phosphorous from bone; it suppresses urinary calcium, causing serum calcium levels to rise and serum phosphate levels to fall. Vitamin D stimulates absorption of calcium and phosphorous from intestinal contents, and accelerates the turnover of both minerals in the bone. General Laboratory Values: 2.5 4.5 mg/dl PHOSPHOROUSIncreased Renal/Liver Dysfx. Ovarian Hyperfunction Fractures; Increased Vit. D levels Decreased Digestive Dysfx; <HCL Ovarian Hypofunction Diabetes; Decreased Vit. D. levels WATER: KIDNEY/BLADDER SODIUM Sodium levels alone are of limited diagnostic use. It is important to compare the relative measurement of sodium vs. potassium. Excessive sodium levels implies heat in the kidney. Low levels of serum sodium imply Kidney Qi Deficiency. General Laboratory Values: 135 147 mEq/L SODIUMIncreased Decreased Adrenal Cortical Hyperfunction Adrenal Cortical Hypofunction Congestive Heart Failure Diabetes; Diarrhea Diabetes; Water softeners Excessive perspiration following exercise POTASSIUM Potassium levels can indicate in which general direction the body pH is going. Increased levels of potassium are indicative of acidosis while decreased levels are indicative of alkalosis. With chronic potassium elevation one has to consider immunodepression and/or inflammation. General Laboratory Values: 3.5 5.3 mEq. per liter POTASSIUMIncreased Adrenal Hypofunction Asthma, Emphysema Renal Dysfunction Decreased Adrenal Hyperfunction Vomiting; Diuretics; Diarrhea Hypertension CHLORIDES Sodium, potassium and chloride ions surround the cell plasma membrane. Comparing the measurements of these three ions one can get general information on the basic functioning of the kidney and the relationship of the water and the earth elements. Generally, elevated chlorides will represent Heat and decreased chlorides will represent Deficiency within the water elements. General Laboratory Values: 96 109 mEq/L CHLORIDESIncreased CO2 deficiency Adrenal Hyperfunction Hyperaparathyroidism Dehydration Salicylate toxicity Renal Dysfunction Decreased CO2 excess Adrenal Hypofunction Respiratory distress Diabetes BLOOD UREA NITROGEN (BUN) Urea is formed almost entirely by the liver from protein metabolism in the tissues. The rate of urea production is accelerated by a diet high in animal proteins and chronic tissue damage. It is believed that more than 50% of the kidney must be destroyed before serum urea levels are significantly elevated. Elevated BUN with normal creatinine usually signals a non-renal cause for uremia. General Laboratory Values: 7 25 mg/dl BUNIncreased Renal Dysfx; Gout; Heart Failure Liv/Biliary Dysfx; Rheum. Arthritis Decreased Celiac Sprue; Advanced Acidosis CREATININE Creatinine clearance is a good measurement of glomerular function. It measures the rate of excretion by the kidneys of metabolically produced creatinine. Blood Creatinine rises when renal function declines. With severe renal impairment, urea levels continue to climb, but creatinine values plateau. General Laboratory Values: .6 1.5 mg/dl CREATININEIncreased Reduced blood flow to kidney Urethral obstruction/stones Shock, Blood Loss, Dehydration, Burns Muscle trauma; Flu; Late pregnancy Decreased not significant URIC ACID Uric Acid is the chief end product of purine metabolism. Purines are constituents of nucleic acids. Most uric acid is synthesized in the liver, in a reaction requiring the enzyme xanthine oxidase. Uric acid travels through the blood to the kidneys, where filtration, absorption and secretion will affect uric acid excretion. Organ meats, legumes and yeast are especially high in purines. Uremia can be described as symptoms and physical abnormalities that result from the kidneys failure to remove nitrogenous waste products normally excreted in the urine. The toxic effects of uremia affect virtually all human organs. The most common symptoms are high blood pressure, swelling (edema) of the ankles, nausea, vomiting and weight loss. Anemia is almost always present because high blood levels of urea, one of the nitrogenous substances, shortens the life span of red blood cells. Other symptoms may include irritation of the heart sac (pericarditis), bleeding, muscle twitches and itching (pruritus). In the later stages, uremia causes agitation alternating with stupor, convulsions, coma, and ultimately death. Analysis of blood chemistries show elevated levels of urea, creatinine, uric acid, phosphorus and hydrogen ion. General Laboratory Values: Males: 3.0 9.0 mg/dl Females: 2.2 7.7 mg/dl URIC ACIDIncreased Decreased Gout, Liv/Kid Dysfx. Pregnancy Rheumatoid Arthritis Anemia B-12 Arteriosclerosis; Heart Disease PROGESTERONE Progesterone is secreted by the corpus luteum of the ovaries and to a lesser extent by the adrenal cortex in both males and females. It prepares the endometrium to received a fertilized ovum and is necessary for the maintenance of early pregnancy. General Laboratory Values: Follicular Phase under 150ng/100ml Luteal Phase: at least 300ng/100ml PROGESTERONEIncreased Benign Prostatic Hypertrophy Pregnancy Decreased Ovarian Tumor/Hypofunction Anterior Pituitary Hypofunction (TSH) OVARIAN HYPOFUNCTION Increased Sodium; Chloride; Cholesterol Decreased Calcium; Phosphorous; Thyroid Binding Globulin FOLLICLE STIMULATING HORMONE (FSH) The ovary secretes estrogens and progesterone under the influence of FSH, which is controlled by hypothalamic releasing factors and stimulated by the pituitary hormones. Estrogens are secreted by ovarian follicular cells in the first half of the menstrual cycle, and by the corpus luteum during the luteal phase and pregnancy. General Laboratory Values: Males: 4 25 mlU/ml Females: 4 30 mlU/ml FSHIncreased Menopause/Post Menopause Decreased Anterior Pituitary Hypofx. (TSH) LUTEINIZING HORMONE (LH) LH and FSH control the function of the hormones of the ovaries and testis. High LH levels inhibit estrogen and stimulate progesterone secretion. General Laboratory Values: Males: 7 24 mlU/ml Females: 6 30 mlU/ml LHIncreased Hyperthyroidism. Polycystic Ovaries Menopause/Perimenopause Decreased Anterior Pituitary Hypofx Benign Prostate Hypertrophy WOOD: LIVER/GALLBLADDER The liver contains complex parenchymal cells which perform multiple diverse functions essential to life. Hepatocytes have the unique ability to regenerate as well as the capacity to respond to increased metabolic demands. The liver directly receives, processes and stores materials absorbed from the digestive tract such as amino acids, carbohydrates, fatty acids, cholesterol and vitamins and is able to release metabolites of these compounds on demand. The liver synthesizes plasma proteins such as albumin, globulin, clotting factors and transport proteins. These factors influence homeostasis, since binding proteins modulate the circulating total concentrations of calcium and magnesium while albumin concentrations regulate osmotic pressure and thus influence the fluid dynamics between the blood and the tissues. The liver is the main organ of detoxification and is the site of metabolic conversion of endogenous and exogenous compounds. The liver also synthesizes bile acids from cholesterol and secretes these compounds from the hepatocytes into the intestine. This generates bile flow and facilitates the emulsification and absorption of fats. The liver is also a major site of catabolism of thyroid, steroid and other hormones. The liver helps to regulate plasma hormone levels. Last, but not least, the liver responds to multiple hormonal and neural stimuli to regulate the blood glucose concentration and contributes to steadying the body immune system. The liver is truly THE GENERAL. LACTIC DEHYDROGENASE (LDH) LDH is found in the blood as well as in all tissues. It represents a group of enzymes involved in carbohydrate metabolism. LDH is an enzyme which assists in the conversion of Pyruvate (pyruvic acid) to lactate or lactic acid in the anaerobic glycolysis of glucose. Lactic acid can be reconverted to pyruvate with the enzymatic assistance of LDH, enter into the mitochondrion and be transformed to ATP for cellular energy. LDH is widely distributed with high concentrations in the heart, musculoskeletal system, liver, kidney, brain and red blood cells. The measurement of total LDH is therefore a non-specific index of cellular damage. General Laboratory Values: Males: 120 240 IU/ml Females: 120 220 IU/ml There are laboratories which do an electrophoresis process which will assess the 5 different isoenzymes of LDH #1 measures heart tissue damage 10 34% of total LDH #2 measures heart, lymph and erythrocyte damage 3 - 45% of total LDH #3 measures pulmonary, spleen, adrenal and kidney 13 27% of total LDH #4 measures hepatic and prostate and uterus 2 14% of total LDH #5 measures hepatic tissue damage 3 15% of total LDH NOTES ABOUT LDH ISOENZYMES: LDH Isoenzyme #1 with an infection and an elevation of total WBC this will be elevated. LDH Isoenzyme #1 with a decreased TSH (associated with thyroid medication) will be mildly elevated (fire/thyroid correlation) LDH Isoenzyme #5 if decreased to less than 6% of the total LDH there may be a history of long term exposure to toxic gases such as leaking gas stoves, etc. A lowered LDH #5 may also be indicative of toxic metal poisoning. LDH INCREASED IN: Any increase in LDH is an implication that there is some tissue damage somewhere in the body. If one chooses to do an electrophoresis Isoenzyme test one can determine which tissue is undergoing a destructive process. LDH DECREASED IN: Sometimes with Hypoglycemia Generally it is believed that increased LDH is representative of excessive acidity and decreased LDH is representative of excessive alkalinity. TOTAL PROTEIN All cells manufacture proteins, different proteins characterizing different cell types. All human proteins are constructed from a mere 20 amino acids, but variations in chain length, amino acid sequence and incorporated constituents combine to make possible an almost infinite number of different protein molecules. Amino acids enter the body from dietary sources. These amino acids are rapidly distributed to tissue cells, which promptly incorporate them into proteins. Protein synthesis and degradation occur continuously, at a rate of approximately 400g. daily. Each day about 20-30g of protein is irreversibly degraded; this is the minimum amount of protein that must be ingested to maintain a metabolic nitrogen balance. General Laboratory Values: 6.0 8.5 g/dl PROTEINIncreased Acidosis; Low HCL Dehydration Amino Acid Def. Decreased Gastritis; Colitis, etc.; Malnutrition Hyperthyroidism ALBUMIN A globular protein which contributes to approximately 60% of the total plasma proteins. It is produced in the liver and is dependent on the intake of amino acids. Albumin binds Bilirubin, free fatty acids and transports and stores numerous metabolic constituents such as thyroxine, cortisol, calcium, magnesium and amino acids. Albumin is responsible for about 80% of the colloid-osmotic pressure between blood and tissue fluids. When albumin is diminished, osmotic pressure is disturbed. A low albumin combined with elevated SGPT and GGTP indicates significant liver dysfunction. General Laboratory Values: 3.5 5.5 g/dl ALBUMINIncreased Hypothyroidism Dehydration Decreased Hyperthyroidism Liver/Kidney Dysfunction; Pregnancy Digestive inflammation; Hypochlorhydria Benign Hypertension; Congestive Heart Failure NOTATIONS: An albumin below 3.5 combined with a lymph count below 20 is indicative of a Free Radical problem. GLOBULIN A globulin is a protein that is insoluble in pure water, in contrast to an albumin which is soluble. Human blood serum contains several globulins that differ in molecular size, amino acid composition, solubility. Total serum globulin increases during recovery from infection. Such beta-globulins as transferring transport iron, heme, and less often, copper and zinc throughout the body. General Laboratory Values: 2.0 3.5 g/dl GLOBULINIncreased Decreased Malnutrition/ Burns; Cirrhosis; Pregnancy Acute Hepatitis/Liver Dysfx.; Congestive Heart Failure SERUM GLUTAMIC OXALOACETIC (SGOT) TRANSAMINASEASPARTATE AMINOTRANSFERASE (AST) SGOT is an enzyme found in the cytoplasm of liver, kidney, myocardial and skeletal muscle cells. Following in injury, infection, inflammation, concentrations of SGOT will > within 10 hours. Within 4-6 days, SGOT levels should resume normal levels. SGOT measurements give information on liver/heart relationship. General Laboratory Values: 1 40 U/l SGOTIncreased Hepatitis/Alcoholism (SGPT, GGTP) Mononucleosis; Flu (WBC) Hypertension; Diabetes (Glucose) Congestive Heart Failure (BUN^ /SGPT^; MCV^; All. Phos^; CO2<; Albumin<) Liver, Kidney, Pancreatic Dysfunction Rheumatoid Arthritis Pericarditis; Myocarditis (LDH>; EOS>/ Neutrophils<) Decreased: B-6 deficiency RX: ELEVATED SGOT Days 1 3: Echinacea Combination dose QID Yin Qiao dose QID Vitamin C dose QID Days 4 7: Echinacea Combination dose BID Isatis Formula dose QID Days 8 15: Minor Bupleurum Formula dose BID Isatis Formula dose BID SERUM GLUTAMIC PYRUVIC TRANSAMINANSEALANINE (SGPT) AMINOTRANSVERASE (ALT) Most concentrated in the liver and in lesser amounts in the kidney, heart and skeletal system. Increased exercise can increase SGOT while SGPT will remain stable. SGPT is principally used to measure liver damage. SGPT is more reliable to measure chronic cellular damage whereas SGOT is more sensitive to acute damage. An individual with viral hepatitis will have a considerably higher SGPT/SGOT than one who has recently had an MI or cancer. (This is in general a measurement of heat and/or stagnation). When using SGPT to dx. Gallbladder problems, remember to test urine and Bilirubin. Often SGPT and SGOT will be normal yet the GGTP will be elevated in gallbladder dx. General Laboratory Values: 2 45 U/l SGPTIncreased Mononucleosis/Influenza Hepatitis (GGTP^; Alk. Phos^; Bilirubin^ Globulin^; Cholesterol<; Mb.<; Platelets<; Albumin<) Alcoholism; Biliary Obstruction (GGTP) Choleycystitis (SED^; WBC^; WBC^; Alk. Phos^ LDH^; Protein^; Bilirubin^; Iron<; Cholesterol<; RBC<; HCT/HGB<;Uric Acid<; Phos.<; Mg.< Acute MI; Myocarditis Renal Dysfunction; Rheumatoid Arthritis Decreased B-6 deficiency ALKALINE PHOSPHATASE A high content of alkaline phosphatase is found in the intestinal mucosa, liver and bone. An elevation of Alk. Phos. is found in both hepatic dysfunction and osteoblastic bone lesions. It is therefore useful in determining relationship between water and wood. An elevated Alk. Phos. in children is most often due to excessive bone growth activity. It is most commonly elevated in Liver Stagnation with biliary tract congestion. Exceedingly low levels of Alk. Phos. are most commonly found in patients with depressed zinc levels. Patients with liver/gallbladder stagnation/obstruction often have higher serum Alk. Phos. than the individual with hepatocellular disorders such as simple Liver Heat. Steroidal drugs and birth control pills will often increase Alk. Phos. General Laboratory Values: 1-9 years: 114-300 U/l 10-14 years 105-350 (M); 58-293 (F) 15-19 years 47-144 (M); 35-109 (F) 19 and above 35-115 U/l ALKALINE PHOSPHATASEIncreased Biliary/liver congestion (SGPT, GGTP) Hepatitis; Mono; Parasites (EOS) Herpes Zoster; Hyperthyroidism Osteoporosis; Rheumatoid Arthritis Congestive Heart Failure (SGOT/LDH); Phlebitis DecreasedHypothyroidism; HypoparathyroidismDigestive Incompetence (protein/fat)Zinc Deficiency; Folic Acid AnemiaHERPES ZOSTERIncreased Alk. Phos.; LDH/ BUN Monocytes; Creatinine DecreasedAlbumin; CholesterolPHLEPITISIncreased DecreasedAlk. Phos; SGOT; WBC Lymphs Eosinophils; Neutrophils RHEUMATOID ARTHRITISIncreased Alk. Phos. Sed. Rate Decreased HCT/HGB WBC GAMMA GLUTAMYL TRANSPEPTIDE (GGPT) GGPT is often the first enzyme to be elevated with liver dysfunction. The elevation continues for as long as hepatic cellular damage persists. It is a sensitive indicator of biliary obstruction. It is interesting to note that elevated Bilirubin in the urine will be evident long before SGOT , SGPT or Alk. Phosphatase become elevated. GGPT is often high with severe alcoholism and correlates with six or more alcoholic drinks daily. General Laboratory Values: 0-65 (M) U/L 0-45 (F) U/L GGPTIncreased Biliary Obstruction; Alcoholism (SGOT;SGPT) Decreasednot significantBILIARY OBSTRUCTIONIncreased GGPT; SGPT; SGOT; Alk. Phos.; WBC Bilirubin; Triglycerides; Cholesterol; LDH Decreased Vitamin A & K levels BILIRUBIN Bilirubin is the end product of the breakdown of hemoglobin by the liver, spleen and bone marrow. The liver transforms Bilirubin to a form that is then excreted through the biliary system or the kidneys. An increase in Bilirubin is responsible for various types of jaundice. Bilirubin helps to rule biliary obstruction in or out. When direct Bilirubin is simultaneously elevated with GGTP and SGPT biliary obstruction is likely. Total Bilirubin is a combination of direct and indirect Bilirubin. General Laboratory Values: Total Bilirubin - .1-1.2 MG/DL TOTAL BILIRUBINIncreased Hepatitis; Mononucleosis Liver/Biliary Dysfunction Decreased Iron Anemia RX: ELEVATED BILIRUBIN Milk Thistle Extract Cholacol Choline Dandelion Extract; Chelidonium Extract; Chionanthus Extract Beet tops; Black Radish TRIGLYCERIDES Triglycerides are esters of glycerol and fatty acids. Elevated triglycerides will often indicate poor utilization of fatty acids. Decreased triglycerides will indicate poor release of fatty acids. Typically, when the triglycerides are elevated there is primarily Damp Heat in the Spleen. When cholesterol is elevated there is more commonly Liver Stagnation. General Laboratory Values: 30-200 MG/DL TRIGLYCERIDESIncreased Liver-Biliary dysfunction Pregnancy; diet ^ in simple sugars/fats Free Radical Pathology Decreased Auto immune dysfunction Protein malnutrition NOTE: When cholesterol and triglycerides are elevated, the possibility of a diet high in simple sugars and fats is most likely. Red meat, alcohol and simple sugars should be restricted when attempting to lower triglycerides. Treatment principle in lowering triglycerides is most often: Harmonize the wood and remove Dampness from the Spleen. The treatment to increase triglycerides is most often: strengthen the Spleen and Wei Qi. RX: TRIGLYCERIDES INCREASED Salvia Combination Wobenzym Burdock Root Extract Guan Jie Formula (w/rheumatoid arthritis) Coptis & Scute Formula followed by Citrus & Crataegus Formula Cardamon & Fennel Formula (w/ stomach pain/ulcer) CHOLESTEROL Cholesterol is a steroid found in all cells in the body. It is synthesized by the liver and often increased in the blood through fat intake. Lipid levels are controlled by several organ systems: kidney; thyroid; adrenals, pituitary and pancreas. Depressed thyroxin levels can crate an increased blood cholesterol by preventing the formation of bile to reduce cholesterol. When the thyroxin level is low with an increased cholesterol, it is important to rule out a pituitary problem. When the pituitary is not secreting sufficient TSH, the thyroid will not be able to produce enough T3 or T4, nor will T3 and T4 be released from the thyroid gland to the blood where these hormones can be activated by estrogen, testosterone and micronutrients. General Laboratory Values: 125 200 mg/dL CHOLESTEROLIncreased Live/Biliary Dysfx. Gout; Lupus; Multiple Sclerosis Diabetes; Arteriosclerosis; Endocrine Dysfx. Chronic Benign Prostate Hypertrophy Pancreatic/Renal/Cardiac Dysfx. Free Radical Pathology Decreased Infection; Hepatitis; Mono Herpes Zoster Protein Malnutrition Immune Deficiency Mental Retardation HDL The only known carrier of cholesterol from peripheral tissue to the liver is HDL. HDL, synthesized by the liver, passes into the bloodstream and takes up excess cholesterol from the cells. A relatively high HDL protects tissue from becoming inundated by cholesterol. General Laboratory Values: 30-75 mg/dL (M) 40-90 mg/dL (F) Musiclear wrote: The liver is responsible for taking potentially harmful compounds and breaking them down into less harmful compounds. It will allow some to reenter the blood, others to be expelled through bile and in some situations, the liver will store them in fat. This is part of the normal process of the liver. Bile flow is an important aspect of the " detox " process otherwise, the liver has trouble getting rid of the compounds and is forced to either allow them back to the blood or store them in fat. If a person has had liver Qi stag, it is my belief that the liver will be holding on to more toxins than if the Qi was moving freely. If a person improves bile and Qi flow or biochemistry, then the liver is more able to resume the job of getting rid of toxins. If there has been an abundance of toxin stored, there may be a lot of toxins dumped into bile or blood which could give a person symptoms that many would call a " Healing Crisis " . IMHO, Chris In a message dated 8/11/2004 3:16:18 AM Eastern Daylight Time, writes: I ran across this post. I hate to be a jerk but what do others think a liver detox is? I'm not coming down on Chris, it's just I hear this all the time and i realized I don't know what it means. doug > Methylation is a key process in helping the liver detox, so given Sam-e > adds some key ingredients to the methylation cycle, it could improve liver > detox function if it has been over worked with toxins and the methylation cycle > has been diminished. > > IMHO, > > Chris Quote Link to comment Share on other sites More sharing options...
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