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A difficult case study: My wife-seizures withParkinsonism, installment #9

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Hi Yehuda

I have a question regarding the pdrecovery. If it tuns out that they are truly

up on to something Real, should we claim it to be part of CM? Since this was

developed by US LAcs is this the so-called property of the acupuncture

profession and now part of CM? Since they think of it as affecting the " St

channel " is this CM? Since the techniques are not what we think of as tuina is

this updated CM bodywork? You get my point.

I wish you wife the best of luck and awaiting updates

Thanks

 

 

 

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Dear Alon,

 

Your questions are certainly valid. First, I again emphasize, PLEASE

read the practitioner handbook updates: www.pdrecovery.org . The entire

basis of the theory behind their work as well as the Yin tuina is all

based upon classical sources, particularly the Nei Jing, as Janice points

out. FSR is merely a translation of Zhi Fa Bu Yong Li Yin Tui Na. Zhi

Fa means to generate by itself, Bu Yong Li means not using force, thus

for simplicity sake Janice called it Forceless Spontaneous Release. She

also points out, interestingly, that in Deadman et al’s “Manual of

Acupuncture”, in the indications for St 42, he translates literally from

the Chinese to “desires to ascend to high places and sing.” At some point

he makes a note that this must be a reference to mental illness.But, to

quote her, ...”There is a better, narrower and more literal possibility;

this seemingly non-medical condition might easily be applied to many

Parkinson’s patient if hte more current, English idiomatic expression,

“desires to climb to the top of the heap and crow’ were substituted.

When this vernacular translation is suggested, the roster of tyrants and

would be emperors with PD immediately springs to mind. While many

patients with PD have been described as “control Freaks” by their friends

and children, and even themselves, the phrase, “desires to ascend to

high places and sing’ says the same thing as “control freak”, but much

more gently and poetically.” So that’s the emotional mindset that I

mentioned in the previous post. Listen to what ”Acupuncture, a

Comprehensive Text” (O’ Connor & Bensky) list as one of the indications

of St 42, “facial paralysis (the parkinson’s mask)...no strength in the

arms and legs (the rebellious Yang Ming Qi in the St and LI),” Anyway,

the point I want to make is that is is not some “out-there,New- age

therapy”, but rather a well researched successful therapy which utilitzes

and is faithful to traditional sources. Furthermore, yin tui na is not

something new, and is as old as tui na, itself closely related to medical

qi gong.

 

Sincerely,

 

Yehuda

 

Hi Yehuda

I have a question regarding the pdrecovery. If it tuns out that they are

truly up on to something Real, should we claim it to be part of CM? Since

this was developed by US LAcs is this the so-called property of the

acupuncture profession and now part of CM? Since they think of it as

affecting the " St channel " is this CM? Since the techniques are not what

we think of as tuina is this updated CM bodywork? You get my point.

I wish you wife the best of luck and awaiting updates

Thanks

 

 

 

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Yehoda

Thanks for the link, I will read the book. Do you know if their treatment

techniques have been actually used in chinese traditions? and by the way

personally i encourage novel development using OM principles and integrative

thinking. Also do you know how many PD patients have been treated so far?

 

 

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Alon,

 

Specifically I don't know if what they do, has ever been done before for

Parkinson's, per se. But then again, Parkinson's, to the best of my

knowledge is a condition originally observed and written about in 1817 by

the Englishman, James Parkinson, which he called " the shaking palsy " , and

is specifically a disease of civilization. That being the case, I would

suspect that the condition was not present except, perhaps in rare cases

of heavy metal toxicity to the ancients. I asked one of the treating

practitioners who told me that they have now successfully treated over

200 PD patients. On their site they list a number of published articles

Janice Walton-Hadlock has written concerning the theory and protocol.

 

Yehuda

 

Yehoda

Thanks for the link, I will read the book. Do you know if their treatment

 

techniques have been actually used in chinese traditions? and by the way

personally i encourage novel development using OM principles and

integrative

thinking. Also do you know how many PD patients have been treated so far?

 

 

 

 

" When you see someone doing something wrong, realize that it was brought

before

you because you did something similar. Therefore, instead of judging

him, judge yourself. "

 

The Baal Shem Tov Hakodesh

 

 

 

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Hi Yehuda

I am reading the book and have to make a comments. She states that neurologists

state that dopamine is a sedative neurotransmitter and that only now they are

waking up to the truth of it being a stimulate. Well i have news for her. The

association of dopamine and excitation has been know for very long time, ever

since thorazine has been used to treat schizophrenia. Second the effects of

dopamine in PD have nothing to do with peripheral actions on muscles and

therefore i do not understand why she is drawing these conclusions regarding

medical hypothesis. Dopamine is used to increase blood pressure, thorazine a

dopamine antagonist is one of the most powerful sedatives we have.

 

She states dopamine increases during the night only in mice well i have news for

her regarding this as well. The activity of dopaminergic neurons has circadian

variation. Dopamine production increases through the night with each cycle of

REM sleep (in humans). The activity at the nigrostriatal terminals is maximal in

the early morning. This nocturnal variation is more marked in young children and

decreases with age. Dopamine activity in nigrostriatal terminals declines

further during the course of the day (as well as with increasing age),

exacerbating symptoms toward evening and with increasing age.

 

She states: PD has no genetic contributions: well

Dopamine is produced from tyrosine by the action of TH, which uses BH4 as a

cofactor. The first rate-limiting step for BH4 synthesis is GCH. This gene in

humans contains 6 exons, and various mutations have been described. These

mutations result in markedly reduced GCH values (2-20%), with a resultant

decrease in dopamine content.

She also states:

" Parkinson's does not appear when dopamine levels are too low. Parkinson's

appears when the adrenals become so exhausted that they can no longer mask the

damage set in motion by the unhealed injury, damage that includes a hibernating

dopamine system. When the exhausted adrenals can no longer mask the problems,

the physical damage, including dopamine dormancy, is exposed " .

 

Why then using fMRI, PATs, and postmortems clearly show the involvement of the

dopamenegic systems? Also adrenal exhaustion is easily measured does she has any

such measurements?

 

As i continue to read i will have more comments. Its hard for me to believe that

having " cured " 200 cases of PD has not received much more mainstream exposure.

 

 

 

 

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Let me clarify one thing that I said in the last post: When I said that

Parkinson's is a disease of civilization, I meant by that, our modern

urban industrial/technological society, which is certainly not conducive

to wellness and spontaneity. Antecdotally, I have been told that the

country with the highest incidence of Parkinson's is England.

Coincidence?

 

Yehuda

 

 

Alon,

 

Specifically I don't know if what they do, has ever been done before for

Parkinson's, per se. But then again, Parkinson's, to the best of my

knowledge is a condition originally observed and written about in 1817 by

the Englishman, James Parkinson, which he called " the shaking palsy " , and

is specifically a disease of civilization. That being the case, I would

suspect that the condition was not present except, perhaps in rare cases

of heavy metal toxicity to the ancients. I asked one of the treating

practitioners who told me that they have now successfully treated over

200 PD patients. On their site they list a number of published articles

Janice Walton-Hadlock has written concerning the theory and protocol.

 

 

 

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Yehoda

She states that the dopamine acts in reverse to adrenaline. In fact it has

different mechanisms which are concentration-dependent and often are synergistic

to adrenaline. They by no means 2 apposing systems and dopamine is not A

parasympathetic neurotransmitter. For example, a low dose dopamine dilates renal

artery, increasing kidney perfusion. A mid-dose stimulates b 2 receptors as

well, resulting in dilation of skeletal bed BV's to decrease systemic

resistance. While high dose stimulate a 1 (as does epinephrine or adrenaline)

receptors and blood vessels constrict, resulting in an overall increased blood

pressure.

 

 

 

 

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Alon,

 

I would strongly suggest that you Email your comments to the author,

Janice Walton Hadlock directly. In the interest of understanding the

dynamics of which we speak, it would behoove you to address the them to

her rather than me. Her Email address is: pdinfo

 

 

Hi Yehuda

I am reading the book and have to make a comments. She states that

neurologists state that dopamine is a sedative neurotransmitter and that

only now they are waking up to the truth of it being a stimulate. Well i

have news for her. The association of dopamine and excitation has been

know for very long time, ever since thorazine has been used to treat

schizophrenia. Second the effects of dopamine in PD have nothing to do

with peripheral actions on muscles and therefore i do not understand why

she is drawing these conclusions regarding medical hypothesis. Dopamine

is used to increase blood pressure, thorazine a dopamine antagonist is

one of the most powerful sedatives we have.

 

She states dopamine increases during the night only in mice well i have

news for her regarding this as well. The activity of dopaminergic neurons

has circadian variation. Dopamine production increases through the night

with each cycle of REM sleep (in humans). The activity at the

nigrostriatal terminals is maximal in the early morning. This nocturnal

variation is more marked in young children and decreases with age.

Dopamine activity in nigrostriatal terminals declines further during the

course of the day (as well as with increasing age), exacerbating symptoms

toward evening and with increasing age.

 

She states: PD has no genetic contributions: well

Dopamine is produced from tyrosine by the action of TH, which uses BH4 as

a cofactor. The first rate-limiting step for BH4 synthesis is GCH. This

gene in humans contains 6 exons, and various mutations have been

described. These mutations result in markedly reduced GCH values (2-20%),

with a resultant decrease in dopamine content.

She also states:

" Parkinson's does not appear when dopamine levels are too low.

Parkinson's appears when the adrenals become so exhausted that they can

no longer mask the damage set in motion by the unhealed injury, damage

that includes a hibernating dopamine system. When the exhausted adrenals

can no longer mask the problems, the physical damage, including dopamine

dormancy, is exposed " .

 

Why then using fMRI, PATs, and postmortems clearly show the involvement

of the dopamenegic systems? Also adrenal exhaustion is easily measured

does she has any such measurements?

 

As i continue to read i will have more comments. Its hard for me to

believe that having " cured " 200 cases of PD has not received much more

mainstream exposure.

 

 

 

 

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Yehuda

She states

Any of us might know of someone who has performed an impossible action such as

fleeing a maniac by running a mile on a broken leg, or winning an eighteen mile

bicycle race with a broken collarbone.

 

>>>>This is know as stress analgesia and is more do to WDR receptors which are

more NMDA related than adrenaline. Sure the sympathetic system is activated but

the analgesic effects are related to above more than adrenaline

 

 

 

 

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Yehuda

She states:

A fascinating study on brain function and susceptibility to addiction in male

primates who become alpha males suggests that their brain switches from dopamine

system -dominant to adrenaline system-dominant. They also switch from addictable

to non-addictable. Even if the primate is supplied with dopamine-enhancing

drugs, his behavior and brain patterns remain those of an adrenaline-dominant

alpha male. See: Nature Neuroscience 5(2): 169-174, 2002, Mike Nader.

 

>>>>>I strongly suggest you read the article your self.

 

 

 

 

 

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Yehuda

While she says " The shift in social standing to an alpha position creates the

neurological shift towards the adrenaline system that is necessary to maintain

constant vigilance, wariness, and an increase in physical strength. This point

is very important to keep in mind for our discussion of Parkinson's disease. "

The study actually states:

" Whereas the monkeys did not differ during individual housing, social housing

increased the amount or availability of dopamine D2 receptors in dominant

monkeys and produced no change in subordinate monkeys. "

 

 

 

 

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The dopamine system, and dopamine itself (in a healthy person), is already up

and ready to go at all times; it is only inhibited to the extent that adrenaline

systems are being used instead

 

>>>>>Why then all stimulants and drugs such as bupropion are both dopaminergic

and adrenergic?

 

 

 

 

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