Antagonism between exogenous hormones and a good response to AP?

[Guest guest]

Hi All,

 

Korean colleagues (Yang et al, 2003) published that steroid + electro-

AP gave BETTER results than steroid alone or electro-AP alone in

experimental spinal cord injury in dogs.

 

See: Jung-whan Yang, Seong-mok Jeong, Kang-moon Seo and Tchi-

chou Nam*. Effects of Corticosteroid and Electro-AP on Experimental

Spinal Cord Injury in Dogs. J. Vet. Sci. (2003), 4(1), 97-101. College of

Veterinary Medicine, Seoul National University, San 56-1 Shillim 9-

dong, Kwanak-gu, Seoul 151-742, Korea; ªCorresponding author: Tchi-

chou Nam College of Veterinary Medicine, Seoul National University

San 56-1 Shillim 9-dong, Kwanak-gu, Seoul 151-742, Korea Tel: +82-2-

880-8680; Fax: +82-2-888-5310, <tcnam] The aim of

this study was to investigate the effects of electro-AP, corticosteroid,

and combination of two treatments on ambulatory paresis due to spinal

cord injury in dogs by comparing therapeutic effects of electro-AP and

corticosteroid. Spinal cord injury was induced in 20 healthy dogs (2.5~7

kg and 2~4 years) by foreign body insertion which compressed about

25% of spinal cord. There was no conscious proprioception, no

extensor postural thrust, and ambulation. Dogs were divided into four

groups ac cording to the treatment; corticosteroid (group A), electro-AP

(group B), corticosteroid + electro-AP (group AB), and control (group C).

Neurological examination was performed every day to evaluate the

spinal cord dysfunction until motor functions returned to normal.

Somatosensory evoked potentials (SEPs) were measured for objective

and accurate evaluations. The latency in measured potentials was

converted into the velocity for the evaluation of spinal cord dysfunctions.

Pain perceptions were normal from pre-operation to 5 weeks after

operation. Recovery days of conscious proprioception in groups A, B,

AB, and C were 21.2±8.5 days, 19.8±4.3 days, 8.2±2.6 days, and

46.6±3.7 days, respectively. Recovery days of extensor postural thrust

in group A, group B, group AB, and group C were 12.8±6.8 days,

13.8±4.8 days, 5.4±1.8 days, and 38.2±4.2 days, respectively. There

were no significant differences between group A and group B. However,

recovery days of group AB was significantly shorter than that of other

groups and that of group C was significantly delayed (p<0.05).

Conduction velocities of each group were significantly decreased after

induction of spinal cord injury on SEPs (p<0.05) and they showed a

tendency to return to normal when motor functions were recovered. It

was considered that the combination of corticosteroid and electro-AP

was the most therapeutically effective for ambulatory paresis due to

spinal cord injury in dogs.

 

The full text is at: http://www.vetsci.org/2003/pdf/97.pdf

 

I have heard that some veterinary colleagues in the west inject small

doses of steroids at the acupoints, and colleagues in Korea do this

routinely. Is this done in human AP practice also?

 

Some AP teachers suggest that steroid use can inhibit or reduce the

efficacy of AP therapy. Some suggest that for best clinical results of AP

therapy,animals that present for treatment while on steroid therapy

prescribed by another practitioner should be weaned off the steroid

gradually before starting AP therapy.

 

Indeed, the idea goes beyond steroids; some teachers say that high

doses of ANY exogenous hormone (for example insulin or thyroid

hormone) suppresses the body's ability to produce its own hormone,

and that an AP therapy that aims to increase production of that

hormone can be inhibited by the exogenous hormone.

 

I am not sure that the widely held idea of an antagonism between

exogenous hormones and a good response to AP is correct in clinical

practice.

 

What do YOU think?

 

Have YOU found that pre-existing or concurrent administration of

steroid, insulin or thyroid hormone interfered with the AP effect?

 

 

 

Best regards,

 

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