eat yer veggies

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http://www.sciencedaily.com/releases/2006/02/060209185153.htm

 

Veggies Contain Chemicals That Boost DNA Repair And Protect Against Cancer

Need another reason to eat your vegetables? New research shows that some of them

contain chemicals that appear to enhance DNA repair in cells, which could lead

to protection against cancer development, say Georgetown University Medical

Center researchers.

 

In a study published in the British Journal of Cancer (published by the research

journal Nature) the researchers show that in laboratory tests, a compound called

indole-3-carinol (I3C), found in broccoli, cauliflower and cabbage, and a

chemical called genistein, found in soy beans, can increase the levels of BRCA1

and BRCA2 proteins that repair damaged DNA.

 

Although the health benefits of eating your vegetables—especially cruciferous

ones, such as broccoli—aren’t particularly new, this study is one of the

first to provide a molecular explanation as to how eating vegetables could cut a

person’s risk of developing cancer, an association that some population

studies have found, says the study’s senior author, Eliot M. Rosen, MD, PhD,

professor of oncology, cell biology, and radiation medicine at Georgetown’s

Lombardi Comprehensive Cancer Center.

 

“It is now clear that the function of crucial cancer genes can be influenced

by compounds in the things we eat,†Rosen says. “Our findings suggest a

clear molecular process that would explain the connection between diet and

cancer prevention.â€

 

In this study, Rosen exposed breast and prostate cancer cells to increasing

doses of 13C and genistein, and found that these chemicals boosted production of

BRCA1, as well as its sister repair protein, BRCA2. Mutations in either of these

genes can lead to development of breast, prostate and ovarian cancers.

 

Since decreased amounts of the BRCA proteins are seen in cancer cells, higher

levels might prevent cancer from developing, Rosen says, adding that the ability

of I3C and genistein to increase production of BRCA proteins could explain their

protective effects.

 

The study was funded by the Susan G. Komen Breast Cancer Foundation and the

National Cancer Institute and co-authors include Drs. Saijun Fan, MD, PhD,

Qinghui Meng, MS, Karen Auborn, PhD, and Timothy Carter, PhD.

 

 

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